Mapping antigenic epitopes of potato virus Y with antibodies
affinity-purified by using overlapping synthetic peptides

Maija Vihinen-Ranta, Reijo Sironen and Matti Vuento

Vihinen-Ranta, M., Sironen, R. & Vuento, M. 1994. Mapping antigenic epitopes
of potato virus Y with antibodies affinity-purified by using overlapping syn-
thetic peptides. Agricultural Science in Finland 3: 207-211. (Department of Bio-
logy, University of Jyväskylä, FIN-40100 Jyväskylä, Finland.)

Synthetic, overlapping peptides representing the entire amino acid sequence of
potato virus Y (PVY) coat protein were used to affinity-purify antibodies from
polyclonal antisera to PVY. In testing the binding of the purified antibodies to PVY
particles, antigenic epitopes were identified. The N-terminal and C-terminal re-
gions of the PVY coat protein were found to contain most of the antigenic epitopes.
The results will facilitate the development of detection methods for PVY based on
synthetic peptides.

Key words: coat protein, Pepscan, plant virus, PVY

Introduction

Mapping of antigenic epitopes of viral coat pro-
teins by systematic immunochemical analysis of
synthetic, overlapping peptides (Pepscan) can pro-
vide information useful in developing immuno-
chemical detection methods for viruses (Geysen
et al. 1984, Shukla et al. 1989). In its standard
form, this technique involves synthesis of a
number of peptides on acrylic-coated polyethyl-
ene pins and testing of these peptides for binding
of anti-viral antibodies. While this method is ex-
tremely well suited for identificationof sequence-
dependent linear epitopes that are targets of diag-
nostic reagents, it has been argued that the anti-
bodies binding to short peptides may not neces-
sarily bind to whole antigen molecules (reviewed

Abbreviations used: BSA = Bovine serum albumin, OA =
Ovalbumin, PBS = Phosphate buffered saline, PVY = Po-
talo vims Y.

by Tribbick et al. 1991) and thus may not in all
cases represent epitopes present on intact anti-
gen. A recently published modification (Tribbick
et al. 1991) of the standard technique overcomes
this problem. In this modified method, synthetic
peptides are used to affinity-purify antibodies
which can then be tested for binding to intact
antigen. We have been interested in the antigenic
epitopes of potato virus Y, an important plant
pathogen. In our previous study carried out by
using the standard Pepscan procedure, we found
the antigenic epitopes of PVY coat protein to be
distributed along the entire amino acid sequence,
including the C-terminal part of the coat protein
(Vuento et al. 1993). However, in an earlier study,
antibodies to potyvirus particles were found to
bind to peptides derived mainly from the N-ter-
minus (residues 1-70, Shukla et al. 1989). There-
fore, it was of interest to study whether antibod-
ies binding to synthetic peptides also would bind
to PVY.

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Material and methods

Purification of PYY (N strain) and production of
polyclonal antibodies against PYY have been de-
scribed earlier (Vuento et al. 1993). The com-
plete set of overlapping hexapeptides (overlap 5
residues) covering the amino acid sequence
(Shukla et al. 1986) of PVY coat protein was
synthesized by using the methodology of Geysen
(Geysen et al. 1984) with reagents (Epitope map-
ping kit) purchased from Cambridge Research
Biochemicals (Cambridge, UK). This methodol-
ogy uses pentafluorophenyl derivatives of N-flu-
orenylmethoxycarbonyl (Fmoc)-protected amino
acids (Atherton and Sheppard 1985). The pep-
tides, 262 peptides in all, were synthesized on
acrylic-coated polyethylene rods attached to poly-
ethylene racks, each holding 96 rods. The dimen-
sions of the racks were such that all the 96 rods
could be fitted in the wells of a 96-well micro-
titer plate.

The binding of anti-PVY antibodies to these
peptides was tested as described by Vuento et
al. (1993). Affinity purification of antibodies from
polyclonal anti-PVY antisera was carried out by
using the method of Tribbick et al. (1991) ex-
cept that instead of using buffers of extremely
acidic or basic pH, we eluted the antibodies from
the peptide pins with urea, a reversible protein
denaturant (Creighton 1993). The procedure was
as follows: antiserum samples were diluted 1:50
in phosphate-buffered saline, pH 7.4, containing
1% w/v bovine serum albumin, 1% w/v ovalbu-
min and 0.1% (v/v) Tween 20. The diluted an-
tiserum was pipetted in aliquots of 200 pi onto
96-well microtiter plates (ImmunoPlate, Nunc,
Denmark). The peptide-containing rods were in-
serted into these wells so that the globular tips of
the rods were fully covered by the solution. The
peptide-containing rods were incubated in the anti-
body solution for 1 hour at 22°C and then washed
3 times with an excess of phosphate-buffered sa-
line (PBS). To achieve elution, 200 pi of a solu-
tion of 8 M urea in PBS was pipetted into the
wells of similar 96-well microtiter plates. The
peptide-containing rods were inserted into the
wells and incubated for 1 h at 22°C, carefully

shaking the plate occasionally. After this time,
the rods were removed, washed with an excess of
PBS, distilled water and methanol, air dried and
stored at room temperature until used again. The
urea solutions containing eluted antibodies were
immediately diluted to neutralize the denaturing
effect of concentrated urea, and analyzed by us-
ing the enzyme-linked immunosorbent procedures
described below.

To test the binding of antibodies to PVY, mi-
crotiter plates (ImmunoPlate, Nunc, Denmark)
were incubated with purified PVY (100 pi of a
solution containing 20 pg/ml of PVY in PBS)
overnight at +4°C. Excessive binding sites were
blocked by incubation with PBS containing 1%
bovine serum albumin and 1% ovalbumin (PBS-
BSA-OA) for 4 hours at 22°C. After thorough
washing with PBS, aliquots of PBS-BSA-OA
(100 pi) were pipetted into the microtiter wells.
Aliquots (10 pi) of antibodies eluted with 8 M
urea were added by mixing to wells containing
the PBS-BSA-OA solution. The microtiter plates
were incubated for 4 hours at 22°C and then
washed thoroughly with PBS. Bound antibodies
were quantitated by incubation with peroxidase-
labelled anti-rabbit IgG antibodies (DAKO, Den-
mark) diluted in PBS-BSA-OA for 2 hours at
22°C. After thorough washing with PBS, peroxi-
dase activity was measured by using a Multiskan
Plus microtiter plate reader (Labsystems, Helsin-
ki, Finland) equipped with a 405-nm filter with
2,2'-azino-bis(3-ethylbenzthiazoline-6-sulfonic
acid) as a substrate. For controls, nonimmune rab-
bit serum was allowed to react with synthetic
peptides, and the material eluted from the pep-
tides was assayed as described above.

Results and discussion

The binding to PVY of antibodies eluted from
synthetic overlapping hexapeptides derived from
PVY coat protein is shown in Fig. la. The immu-
nodominance of the N-terminal and C-terminal
parts of the amino acid sequence is indicated by
the frequency of reacting antibodies (Fig. la).
The frequency of reacting antibodies was higher

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at the N-terminus than at the C-terminus. How-
ever, antibodies binding to PVY were also eluted
from peptides derived from the central part of the
amino acid sequence. The present results reflect
the distribution of linear epitopes only since pos-
sible non-linear epitopes would not show up by
this approach. We also recognize that binding of
viral particles to plastic surfaces may result in
unfolding of the particles (Laver et al. 1990) and
in modification of epitopes of native antigens
(Stevens et al. 1986). Despite these limitations,
the results strongly support the model of Shukla
et al. (1988), suggesting that the N-terminus and
the C-terminus of the amino acid sequence are
located at the surface of the PVY coat protein
structure and induce antibodies during immuni-
zation. This suggestion is also supported by find-
ings that immunization with peptides derived from
N-terminal and C-terminal regions has given rise
to antibodies that react with PVY (Ohshima et

al. 1992, Vuento et al. 1993). It appears from
the present data that some parts of the central
region may also have similar properties.

Comparison of the present data with results
obtained by the standard approach of measuring
the binding of anti-PVY-antibodies to peptides
(Fig. lb) revealed a few interesting differences.
In a few cases, antibodies binding to peptides
gave only weak signals when eluted and tested
for binding to PVY. These cases included the
peptides 85-89, 185-186, 241-247, 256 and 261-
262. The antibodies binding to these peptides ap-
parently did not recognize the respective epitopes
in the viral particles. The amino acid sequence
(PVY strain D, Shukla et al. 1986) used for syn-
thesis of hexapeptides varies in a few amino acid
residues from the corresponding amino acid se-
quence of the viral strain (PVY strain N) used
for immunization. The number of variant amino
acid residues has been reported to be 9 (Robaglia

Fig. la. Binding of affinity-purified antibodies to PVY. Antibodies were purified from anti-PVY
antisera by using a complete set of synthetic, overlapping hexapeptides covering the entire amino acid
sequence of PVY coat protein. The purified antibodies were assayed for binding to PVY with the
enzyme-linked immunosorbent assay (ELISA). Signals (A405 ) obtained in ELISA are shown on the
vertical axis. Signals given by non-immune rabbit serum have been subtracted from the data shown.
Peptides are identified by the number (counted from the N-terminus of the amino acid sequence) of
their first amino acid residue.

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Agricultural Science in Finland 3 (1994)



et al. 1989) or 20 (van der Vlugt et al. 1989),
apparently depending on the virus isolate used
for analysis. However, this variation could not
explain the above results since the variant sites
were not included in the above mentioned pep-
tides.

On the other hand, the present experiments
identified as epitopic several peptides not detect-
ed by the standard method of testing binding of
anti-PVY antibodies to peptides. Thus the pep-
tides 23-28, 36-38, 42-43, 55-56 and 200-202
did not give signals in the direct assay (Fig. lb);
yet antibodies reacting with PVY were eluted from
these peptides (Fig. la). It has been suggested, in
connection with a similar case (Tribbick et al.
1991), that even low amounts of antibody, escap-
ing detection when bound to peptide, may have
after elution sufficient affinity to show signifi-
cant binding to viral antigens. In the case of pep-
tides 23-28, 36 and 55-56, but not in the case of

peptides 37-38, 42-43 and 200-202, the amino
acid substitutions (at positions 24, 26, 29, 31, 36
and 58; Robaglia et al. 1989, van der Vlugt et
al. 1989) between peptides and the virus may
have contributed to the weakness of binding of
an anti-PVY antibody to a peptide. Taken togeth-
er, these results suggest that in order to obtain a
reliable epitope map of a protein antigen, one
should test both for antibodies bound to peptides
and for antigen-binding antibodies eluted from
the peptides. The mapping of antigenic epitopes
of PVY coat protein will facilitate the develop-
ment of detection methods for PVY, based on
synthetic peptides and antibodies to synthetic pep-
tides. This work is currently in progress in our
laboratory.

Acknowledgements. This work was supported in part by
the Council for Agricultural Sciences, Academy of Fin-
land. We thank Ms. Pirjo Kauppinen for skilful technical
assistance.

Fig. lb. Pepscan analysis of binding of anti-PVY antibodies to synthetic peptides. The set of peptides
used and the polyclonal anti-PVY antiserum were the same as in Fig. la. The peptides binding
antibodies from the anti-PVY antisera are identified as vertical columns (the scale is arbitrary). All
peptides giving an ELISA signal at least twice the size of the background signal (background signals
were determined by using non-immune rabbit serum) have been included.

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Manuscript received February 1994

SELOSTUS

Perunavirus Y:n kapsidiproteiinin antigeenisten epitooppien kartoitus käyttäen
synteettisten peptidien avulla affiniteettipuhdistettuja vasta-aineita

Maija Vihinen-Ranta, Reijo Sironen ja Matti Vuento

Jyväskylän yliopisto

Työssä syntetisoitiin limittäisten heksapeptidien sarja, joka
kattoi perunavirus Y:n (PVY) kapsidiproteiinin koko ami-
nohapposekvenssin. Peptidien avulla affiniteettipuhdistet-
tiin anti-PVY vasta-aineita polyklonaalisista anti-PVY an-
tiseerumeista, ja vasta-aineiden sitoutuminen PVY-partik-
keleihin tutkittiin entsyymi-immunoassaymenetelmillä.
Näin saatiin identifioitua PVY:n kapsidiproteiinin anti-
geenisiä alueita, joita löydettiin erityisesti kapsidiproteii-
nin polypeptidiketjun päistä. Vertailu menetelmään, jossa

mitattiin anti-PVY-vasta-aineiden sitoutumista ko. pepti-
disarjan peptideihin, paljasti useita eroavaisuuksia.

Tulokset osoittavat, että luotettavan epitooppikartan saa-
miseksi on tutkittava sekä vasta-aineiden sitoutumista pep-
tideihin että peptideistä eluoitujen vasta-aineiden sitoutu-
mista virusantigeeniin. Saadut tutkimustulokset auttavat
synteettisiin peptideihin perustuvien määritysmenetelmien
kehittämisessä kasviviruksille.

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Agricultural Science in Finland 3 (1994)