Archives of Academic Emergency Medicine. 2020; 8(1): e73

OR I G I N A L RE S E A RC H

Usefulness of Immature Granulocytes to Predict High
Coronary SYNTAX Score in Acute Coronary Syndrome; a
Cross-sectional Study
Cihan Bedel1∗, Mustafa Korkut1, Fatih Aksoy2, Görkem Kuş3

1. Department of Emergency Medicine, Health Science University Antalya Training and Research Hospital, Antalya,Turkey.

2. Department of Cardiology, Suleyman Demirel University Faculty of Medicine, Isparta, Turkey.

3. Department of Cardiology, Health Science University Antalya Training and Research Hospital, Antalya,Turkey.

Received: July 2020; Accepted: August 2020; Published online: 15 September 2020

Abstract: Introduction: Immature granulocytes (IG) in peripheral blood indicate increased bone marrow activation and
inflammation, and SYNTAX score (SS) is an anatomical scoring system based on coronary angiogram. This study,
aimed to evaluate the relationship between IG and SS, as a new inflammatory marker in patients with acute
coronary syndrome (ACS). Methods: Patients aged >18 years who were diagnosed with ACS in the emergency
department were included in this study, which was planned as a cross-sectional study. Patients were divided
into two groups of patients with high and low SSs according to coronary angiography results. Demographic
and laboratory parameters were compared between the groups. Results: Our study consisted of 78 patients
diagnosed with ACS, who met the inclusion criteria. The average age of the study group was 59 years, and 67.9%
of the patients were male. 21 patients (26.9%) had high SSs and 57 patients (73.1%) had low SSs. Mean IG% was
significantly higher in high SS group compared to low SS group (0.71±0.25 vs 0.44±0.21 mg/dl, p<0.001). IG%
can present a high SS with 76.2% sensitivity and 75.4% specificity at a cut-off value of 0.7. Conclusion: IG was
significantly higher in ACS patients with high SSs. It seems that IG can be used as a parameter, which is quickly
accessible and cheap, in order to predict high SS in ACS patients in daily clinical practice.

Keywords: Inflammation; Acute coronary syndrome; Granulocytes; percutaneous coronary intervention; Emergency
Medicine; Atherosclerosis

Cite this article as: Bedel C, Korkut M, Aksoy F, Kuş G. Usefulness of Immature Granulocytes to Predict High Coronary SYNTAX Score in Acute

Coronary Syndrome; a Cross-sectional Study. Arch Acad Emerg Med. 2020; 8(1): e73.

1. Introduction

Acute coronary syndrome (ACS) is one of the main reasons

for admission to the emergency department and hospitaliza-

tion. ACS is usually characterized by atherosclerotic plaque

rupture and complete or incomplete thrombosis of the coro-

nary arteries, which is one of the most significant causes of

mortality and morbidity [1, 2]. Many pathophysiological fac-

tors influence this atherosclerotic process, and inflammation

is one of these factors. Inflammation plays a significant role

in initiating atherosclerosis and facilitating its progression

∗Corresponding Author: Cihan Bedel; Health Science University Antalya
Training And Research Hospital, Kazim Karabekir Street postal zip code: 07100,
Muratpaşa, Antalya, Turkey. Tel: +905075641254, Fax: +902422494487, Email:
cihanbedel@hotmail.com.

[3].

Inflammatory markers, such as white blood cell (WBC), C-

reactive protein (CRP), Neutrophil-lymphocyte ratio (NLR),

and platelet-lymphocyte ratio (PLR), have been researched

in the demonstration of poor condition in cardiovascular

events [4, 5]. Immature granulocyte (IG), a parameter that

is not adequately known by many clinicians, reflects the frac-

tion of immature granulocytes in the peripheral blood. This

parameter can be easily and quickly measured in automated

blood cell analyzers. It has been revealed to be useful in pre-

dicting the severity of many disease processes, such as bac-

terial infection, acute inflammatory diseases, tissue necrosis,

and acute transplant rejection, in recent years [6, 7].

Synergy between percutaneous coronary intervention with

taxus and cardiac surgery (SYNTAX) score (SS) is an anatom-

ical scoring system based on coronary angiogram, which

can help with evaluating the severity of coronary artery

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C. Bedel et al. 2

disease and making revascularization decisions [8]. Previ-

ously, many studies have examined the relationship between

WBC, CRP, Neutrophil-lymphocyte ratio (NLR), and platelet-

lymphocyte ratio (PLR) and SS; however, there are no studies

evaluating the relationship between IG and SS in the litera-

ture. Therefore, this study aimed to evaluate the relationship

between IG and SS, as a new inflammatory marker in patients

with ACS.

2. Methods

Patients aged >18, who had presented to the emergency de-

partment with pain in the chest and been admitted to the

department of cardiology with a diagnosis of ACS (unstable

angina pectoris/myocardial infarction without ST-segment

elevation (NSTEMI)/myocardial infarction with ST-segment

elevation (STEMI)), were included in this study, which was

planned as a prospective cross-sectional study and per-

formed during the period between December 01, 2019, and

February 01, 2020. The diagnosis of ACS was defined as hav-

ing electrocardiographic (ECG) change and/or increase of

cardiac markers along with chest pain, which was assumed to

be typical chest pain. In compliance with American College

of Cardiology and European Society of Cardiology (ACC/ESC)

criteria, STEMI was defined as ST-segment elevation in ECG

and increase in all the derivations by âL’ě0.1mV in two con-

secutive derivations. Necessary approval was received from

the Clinical Research Ethics Committee for the study (No:

IRB-2019-355). Written informed consent was obtained from

all the patients, who agreed to participate in the study.

2.1. Participants

Exclusion Criteria were determined as being <18 years

old, being pregnant, having a myeloproliferative disease (it

may change hematological parameters), malignancy, having

trauma or surgery history within the past 1 week, arrhyth-

mia causing hemodynamic instability, heart failure, inflam-

matory bowel disease, granulocyte-colony stimulating factor,

and using immunosuppressive agents or steroids.

2.2. Data gathering

ACS patients’ age, gender, history of hyperlipidemia, his-

tory of hypertension (HT), history of diabetes mellitus (DM),

family history, drugs, systolic blood pressure (SBP), diastolic

blood pressure (DBP), and heart rates (HR) (pulse/min) were

recorded. The minimum sample size with a two-sided alpha

value of 5%, a statistical power of 80% was estimated to be

50 patients. We planned to enrol a total of 80 patients, taking

into account the 20% expected failure rate.

2.3. SYNTAX Score and Angiographic Analysis

In the study, coronary angiography (CAG) was carried out

for all the patients using the Judkins technique. In order to

grade the stenosis of the coronary vessels, stenoses over 50%

in vessels with a size of ≥1.5mm were taken into consider-
ation. SS was prospectively calculated by two experienced

cardiologists using an algorithm based on the diagnostic an-

giogram. The final score was calculated using individual le-

sion scores by analysts who were blind to operational data

and clinical outcomes. Items such as whether the stenosis

was total, the level and the size of the stenosis, presence of

collateral flow, presence of bifurcation or trifurcation lesion,

severe folds, and severe calcification were evaluated [9, 10].

2.4. Blood Samples

Venous blood samples of the patients were taken within the

first hour of admission to the emergency department be-

fore the primary CAG. In the samples taken during admis-

sion, WBC, neutrophil count, lymphocyte count, and IG% IG

count (IGC) were measured using an automated blood anal-

ysis system (CoulterÂő LH 780 Hematologic Analyzer, Beck-

man Coulter Inc. Brea, USA). Absolute cell numbers were

used in the analyses. CRP, haemoglobin, glucose values,

and cardiac Troponin T levels, which were measured dur-

ing the admission, were recorded. The levels of total choles-

terol, high-density lipoprotein (HDL), low-density lipopro-

tein (LDL), and triglycerides were recorded during admission

to the coronary intensive care. The left ventricular ejection

fraction (LVEF) of the patients was measured using Vivid S5

(GE Healthcare, Inc. Chicago, IL, USA) device connected to

2-4 MHz transducer via Simpson’s method according to the

recommendations of the American Society of Echocardiog-

raphy [11]. According to CAG results, patients with high SSs

(>22) and patients with low SSs (≤22) were separated into two
groups, and all the parameters were compared.

2.5. Statistical Analysis

Statistical analyses were conducted using SPSS 21.0 package

program (SPSS Inc., Chicago, IL). Continuous variables were

expressed as mean ± standard deviation, and categorical
variables were given as number and percentage. An indepen-

dent t-test was used for comparing the distribution of the pa-

rameters with normal distribution, and the Mann-Whitney U

test was applied for those that did not have a normal distribu-

tion. In categorical data, the evaluation was made using the

chi-square test. Logistic regression was conducted for factors

associated with high SS. The optimum cut off value of IG in

predicting high SS was assessed through Receiver operating

characteristic (ROC) analysis. Statistical significance was de-

fined as a p-value less than 0.05.

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3 Archives of Academic Emergency Medicine. 2020; 8(1): e73

Table 1: Comparing the baseline characteristics of patients with high (> 22) and low (≤ 22) SYNTAX score

Variables SYNTAX Score P
High (n=21) Low (n =57 )

Age (years)
Mean ± SD 66.00 ± 16.67 57.57 ± 14.15 0.057
Gender n (%)
Male 15 (68.2) 38 (66.6) 0.789
Female 6 (31.8) 19 (33.4)
Underlying comorbidity
Yes 25.55 34.19 13.62
No 8.71 11.49 5.84
Vital signs
SBP, mm Hg 131.57 ± 14.92 144.14 ± 27.01 0.071
DBP, mm Hg 83.19 ± 8.04 91.47 ± 15.26 0.006
Heart rate, beats/min 83.07 ± 15.26 84.33 ± 15.26 0.283
Ejection fraction, % 45.95 ± 12.57 55.7 ± 12.93 0.002
SYNTAX score 27.78 ± 4.79 7.38 ± 6.24 <0.001
Previous history
Current smoker 12 (57.1) 29 (50.9) 0.799
Hypertension 11 (52.4) 28 (49.1) 0.500
Diabetes mellitus 7 (33.3) 22 (38.6) 0.794
Dyslipidemia 9 (42.9) 21 (36.8) 0.409
History of CAD 8 (38.1) 6 (10.5) 0.009
Laboratory findings
WBC count (×103 /mm3 ) 13.03 ± 3.05 10.68 ± 4.15 0.003
Neutrophil, (×103 /mm3 ) 8.77 ± 2.84 6.84 ± 3.4 0.005
Lymphocyte, (×103 /mm3 ) 3.67 ± 3.20 2.97 ± 2.41 0.318
NLR 4.09 ± 3.91 3.14 ± 2.71 0.367
PLR 112.14 ± 59.08 120.51 ± 57.22 0.499
Hemoglobin, mg/dL 13.89 ± 1.97 13.55 ± 1.97 0.355
Glucose (mg/dl) 163.33 ± 71.52 140.36 ± 69.22 0.017
IGC(×103 /mm3 ) 0.08 ± 0.06 0.07 ± 0.01 0.004
IG% 0.71 ± 0.25 0.44 ± 0.21 <0.001

CRP (mg/dL) 35.37 ± 17.12 3.92 ± 0.54 0.021
Troponin T (ng L) 502.00 ± 157.07 426.08 ± 157.74 0.012
Lipid profiles (mg/dl)
Triglycerides 162.72 ± 92.30 212.33 ± 134.39 0.188
Total cholesterol 211.27 ± 65.80 220.64 ± 56.83 0.304
High-density lipoprotein 44.00 ± 9.01 46.28 ± 10.97 0.354
Low-density lipoprotein 135.50 ± 57.34 134.66 ± 45.95 0.650
Previous medication n (%)
RAS blocker 2 (9.5) 5 (8.8) 0.611
ACE-I 2 (9.5) 14 (24.6) 0.210
Beta blocker 3 (14.3) 15 (26.3) 0.368
Diuretic 4 (19) 9 (15.8) 0.740
Calcium channel blocker 6 (28.6) 8 (14) 0.184
Statin 10 (47.6) 22 (38.6) 0.605
Antiaggregant 5 (23.8) 12 (21.1) 0.766
Oral antidiabetic drug 4 (19) 16 (28.1) 0.562
Mortality
Number (%) 3 (14.3) 0 (0) 0.017
Data are presented as mean ± standard deviation or frequency (%). SYNTAX score: Synergy between percutaneous coronary intervention
with taxus and cardiac surgery; SBP: Systolic Blood Pressure; DBP: Diastolic Blood Pressure; CAD: Coronary artery disease;
WBC: white blood cell; NLR: neutrophil lymphocyte ratio; PLR: platelet lymphocyte ratio; IGC: Immature granulocyte count;
IG%: Immature granulocyte percentage; CRP: C-reactive protein; RAS: Renin–angiotensin system;
ACE-I: Angiotensin converting enzyme inhibitor.

3. Results

Our study consisted of 78 patients diagnosed with ACS, who

met the inclusion criteria. The average age of the study

group was 59 ± 15.21 years, and 67.9% of the patients were
male. 21 patients (26.9%) had high SSs (>22) and 57 pa-

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C. Bedel et al. 4

Table 2: Comparing the baseline characteristics of patients with high (> 0.6) and low (≤ 0.6) immature granulocyte

Variables Immature granulocyte P
High (n=22) Low (n =56 )

Age (years)
Mean ± SD 61.13 ± 20.28 59.33 ± 12.92 0.807
Gender n (%)
Male 15 (68.2) 38 (66.6) 0.601
Female 6 (31.8) 19 (33.4)
Vital signs
SBP, mm Hg 129.45 ± 16.31 145.19 ± 26.37 0.07
DBP, mm Hg 83.54 ± 9.18 91.48 ± 15.15 0.024
Heart rate, beats/min 84.81 ± 18.21 82.85 ± 8.92 0.367
Ejection fraction, % 50.90 ± 11.51 54.01 ± 10.51 0.234
SYNTAX score 17.21 ± 14.80 10.97 ± 8.31 0.024
Previous history
Current smoker 13 (59.1) 28 (50) 0.615
Hypertension 10 (45.5) 29 (51.8) 0.802
Diabetes mellitus 7 (31.8) 22 (39.3) 0.610
Dyslipidemia 5 (22.7) 25 (44.6) 0.120
History of CAD 5 (22.7) 9 (16.1) 0.522
Laboratory findings
WBC count (×103 /mm3 ) 12.88 ± 3.92 10.70 ± 3.90 0.032
Neutrophil, (×103 /mm3 ) 9.26 ± 3.78 6.61 ± 2.87 0.003
Lymphocyte, (×103 /mm3 ) 3.70 ± 0.82 2.97 ± 0.96 0.526
NLR 4.22 ± 4.11 3.06 ± 2.73 0.111
PLR 112.78 ± 61.46 119.95 ± 56.66 0.560
Hemoglobin, mg/dL 12.95 ± 2.25 13.89 ± 1.78 0.131
Glucose (mg/dl) 114.38 ± 63.50 148.07 ± 73.39 0.556
CRP (mg/dL) 24.71 ± 14.98 7.35 ± 3.01 0.693
Troponin T (ng L) 519.66 ± 156.17 424.96 ± 160.55 0.011
Lipid profiles (mg/dl)
Triglycerides 175.11 ± 107.80 208.35 ± 132.05 0.381
Total cholesterol 189.22 ± 41.98 227.73 ± 60.65 0.012
High-density lipoprotein 42.38 ± 8.90 46.80 ± 10.84 0.011
Low-density lipoprotein 118.77 ± 31.18 140.03 ± 52.11 0.08
Previous medication n (%)
RAS blocker 2 (9.1) 5 (8.9) 0.561
ACE-I 3 (13.6) 13 (23.2) 0.535
Beta blocker 4 (18.2) 14 (25) 0.766
Diuretic 4 (18.2) 9 (16.1) 1.000
Calcium channel blocker 5 (22.7) 9 (16.1) 0.522
Statin 6 (27.3) 26 (46.4) 0.135
Antiaggregant 5 (22.7) 12 (21.4) 1.000
Oral antidiabetic drug 2 (9.1) 18 (32.1) 0.45
Mortality
Number (%) 3 (13.6) 0 (0) 0.02
Data are presented as mean ± standard deviation (SD) or frequency (%). SBP: Systolic Blood Pressure; DBP: Diastolic Blood Pressure;
SYNTAX score: Synergy between percutaneous coronary intervention with taxus and cardiac surgery;
WBC: white blood cell; NLR: neutrophil lymphocyte ratio; PLR: platelet lymphocyte ratio; IGC: Immature granulocyte count;
CAD: Coronary artery disease; WBC: white blood cell; NLR: neutrophil lymphocyte ratio; PLR: platelet lymphocyte ratio;
IGC: Immature granulocyte count; IG%: Immature granulocyte percentage; CRP: C-reactive protein; RAS: Renin–angiotensin system;
ACE-I: Angiotensin converting enzyme inhibitor.

tients (73.1%) had low SSs (≤22). There was no statisti-
cal difference between the groups in terms of age and gen-

der (p>0.05). Patients with high SSs had significantly lower

DBP and LVEF (p=0.006, p=0.002, respectively). Patients with

high SSs had significantly higher CAD history comorbidity

(p=0.009). Mean WBC, neutrophil, glucose, IGC, and CRP

and troponin T levels were significantly higher in patients

with high SSs. The mean IG% was significantly higher in the

high SYNTAX score group compared to the low SYNTAX score

group (0.71±0.25 vs 0.44±0.21mg/dl, p<0.001) (Figure 1). Be-

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5 Archives of Academic Emergency Medicine. 2020; 8(1): e73

Table 3: Screening performance characteristics of immature granulocyte percentage in predicting the SYNTAX (synergy between percuta-

neous coronary intervention with taxus and cardiac surgery) score in 0.7 cut off point

Characters Value (95% CI) Characters Value (95% CI)
Sensitivity 76.2 (67.21-84.67) NPV 76.5 (69.15 – 82.59
Specificity 75.4 (66.11-83.81) PLR 3.17 ( 2.2 - 4.57)
PPV 76 (68.74 - 82.02) NLR 0.31 (0.21 –0.45)
Data are presented as mean ± standard deviation (SD) or frequency (%). SBP: Systolic Blood Pressure; DBP: Diastolic Blood Pressure;
Confidence interval (CI). PPV: Positive predictive value; NPV: Negative predictive value;
PLR: Positive likelihood ratio; NLR: Negative likelihood ratio.

Figure 1: Comparison of immature granulocyte levels between low

and high syntax score groups.

Figure 2: Receiver operating characteristic (ROC) curve of imma-

ture granulocyte percentage for predicting high syntax (synergy be-

tween percutaneous coronary intervention with taxus and cardiac

surgery) score (p < 0.001).

sides, the percentage of mortality was significantly higher in

those with high SSs (p=0.017). The demographic data and

laboratory results of the groups are compared in Table 1. 22

(28.2%) patients had high IG% values (>0.6), and 56 (71.8%)

patients had low IG% values (≤0.6). As seen in Table 2, the
prevalence of oral antidiabetic drug use was higher at high

IG% values, and mean DBP, HDL, and LDL values of pa-

tients were found to be significantly lower. In the group with

high IG% levels, higher SSs were detected compared to the

patients with low IG% values (17.21±14.80 vs. 10.97±8.31,
p=0.024). Additionally, in the analysis of the ROC curve, IG%

was shown to predict high SSs with 76.2% (95% Cl: 67.21 –

84.67) sensitivity, 75.4% (95% Cl: 66.11 – 83.81) specificity,

and area under the ROC curve of 0.803 (95% CI: 0.699 - 0.908)

at a cut-off value of 0.7 (Table 3, Figure 2).

4. Discussion

To the best of our knowledge, this is the first study in the lit-

erature evaluating the relationship between IG and SS in ACS

patients. The main findings of this study suggested that SS

was independently correlated with IG%.

SS is a scoring system used to evaluate the complexity and

prevalence of coronary artery disease based on CAG. It is

commonly used by many physicians to specify the opti-

mal cardiovascular treatment strategy [12, 13]. Studies have

shown that patients with high SSs may have poorer cardio-

vascular outcomes, and the score may be an independent

predictor for percutaneous interventions. Moreover, high-

risk patients can be identified using this scoring system, and

appropriate treatment methods can be selected [14, 15].

Inflammation is critically important for the initiation and

progression of coronary atherosclerosis. Inflammation af-

fects many conditions, such as endothelial dysfunction,

leukocyte recruitment, and platelet activation during the

atherosclerosis process [16]. Recently, it has been re-

vealed that many inflammatory markers, such as CRP,

platelet/lymphocyte ratio (PLR) and neutrophil/lymphocyte

ratio (NLR), WBC, TNF-α, and cytokines can be independent

risk factors for atherosclerosis. The increase in these inflam-

matory markers has been shown to correlate with the degree

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C. Bedel et al. 6

and severity of CAD [14, 17]. These inflammatory markers

have been evaluated as prognostic markers for many car-

diovascular diseases, such as coronary artery ectasia, stable

CAD, and myocardial infarction [18, 19]. 840 patients, who

underwent coronary angiography for CAD evaluation, were

included in a recent study by Sahin et al. In this study, NLR

was shown to be significantly associated with CAD severity

in patients with STEMI, and they also reported that NLR is

an independent marker for SS [2]. In a recent study by Al-

tun et al., Troponin T and NLR were significantly associated

with the angiographic severity of ACS evaluated with SS [16].

In a study conducted by Kundi et al., it was reported that the

ratio of the Monocyte count to HDL could be used as a pa-

rameter that would be quickly accessible and cheap in or-

der to predict high SS and it may be used in daily practice

as well [13]. In a study conducted by Sivri et al. on 175 pa-

tients, the WBC/mean platelet volume ratio was shown to

be correlated with increased SS, and thus, short- and long-

term mortality [1]. IG in peripheral blood indicates increased

bone marrow activation, and it can be easily measured in au-

tomated blood analyzers. It has been shown in studies that

the presence of immature granulocytes in peripheral blood,

which is not normally observed in healthy people, can indi-

cate bone marrow activation and serious infection [20, 21].

Recent studies suggest that IG is correlated with prevalent in-

travascular coagulation and mortality in critical patients with

suspected sepsis [22]. Park et al. reported that high IG values

are a good diagnostic sign for severe sepsis and septic shock

within the first 24 hours after admission to the intensive care

unit [23]. Mathews et al. discovered that the increase in IG%

was significant in appendicitis complications in the pediatric

age group and only compared it with an increased CRP level

and left shift [24]. In this study, we showed that patients with

high IG levels had higher SSs. Besides, mortality was higher

at high IG levels.

5. Limitations

The first limitation was that our study, although it was de-

signed prospectively, was conducted with a small number of

patients due to the COVID-19 pandemic. Furthermore, the

mono-center design of our study increases bias. As another

limitation, the fact that the decision on CAG was not made

by the same physicians may have influenced the results. Ad-

ditionally, the period from the emergence of the symptoms

until hospital admission could not be assessed, which may

affect the values of inflammatory markers. Finally, since pa-

tients with a history of CABG were not included in the study,

SS could not be confirmed in this population.

6. Conclusion

IG was significantly higher in ACS patients with high Syntax

scores. It seems that IG can be used as a parameter, which

is quickly accessible and cheap, in order to predict the high

SYNTAX score in ACS patients in daily clinical practice.

7. Declarations

7.1. Acknowledgements

The authors would like to thank MD. Asli BEDEL for helping

in preparation of this paper.

7.2. Author contribution

All the authors have a substantial contribution in the study

design, data interpretation and writing and reviewing the

manuscript.

Authors ORCIDs
Cihan Bedel: 0000-0002-3823-2929

Mustafa Korkut: 0000-0003-1665-1601

Fatih Aksoy: 0000-0002-6480-4935

Görkem kuş: 0000-0002-6058-5501

7.3. Funding/Support

No funding and support was received for this study.

7.4. Ethical approval

Ethics committee approval was received for this study.

7.5. Conflict of interest

The author(s) declared no potential conflicts of interest with

respect to the research, authorship, and/or publication of

this article.

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	Introduction
	Methods
	Results
	Discussion
	Limitations
	Conclusion
	Declarations
	References