id	author	title	date	pages	extension	mime	words	sentence	flesch	summary	cache	txt
biotropia-195	AMPARADO, EDNA A	HISTOPATHOLOGY OF THE TELENCEPHALON AND DIENCEPHALON OF T1LAPIA NILOTICA EXPOSED TO SUBLETHAL DOSE OF MALATHION S-[1,2-DI-(ETHOXYCARBONYL ETHYL) DIMETHYL PHOSPHOROTHIOLOTHIONATE]	2011	12	.pdf	application/pdf	2448	189	53	Necrosis is attributed to the binding of the toxicant to the SH group in the neuronal membrane (Trump et al. 1978), failure of the neuronal plasma membrane ATPase system (Chang et al. 1982) and interaction of the lipophilic pesticide with the cell membrane (Triebskorn and Kunast 1990), and increase in acetylcholine release that induced increased membrane permeability to sodium, potassium, and calcium ions (Kabayashi et al. 1988). Membrane deformations are similar to the necrosis in the brain of Pacific herring larvae, Clupea harenguipallasi, exposed to crude oil (Cameron and Smith 1980), larvae hatched from eggs that have been incubated in varying concentrations of zinc (Somasundaram et al. 1984) and in Paraclithys lethosigma exposed to mercury (Trump et al. 1975) and mouse brain exposed to the neurotoxicant trimethyltin (Chang et al. 1982).	cache/biotropia-195.pdf	txt/biotropia-195.txt