.


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ReseaRch aRticle

Investigation of the Zinc Oxide Nanoparticles Effect on Thyroid 
and Testosterone Hormones in Male Rats
Noori M. Luaibi,* Noor A. Zayed

Department of Biology, College of Sciences, University of Al-Mustansiriyah, Baghdad, Iraq

ABSTRACT

Exposure to zinc oxide nanoparticles (ZnO NPs) has been increasing steadily, causing more attention being paid to their potential 
toxicity, including cytotoxicity and genotoxicity. Hence, this study aimed to investigate the effect of ZnO NPs on thyroid hormone 
triiodothyronine (T3), thyroxine (T4), and thyroid-stimulating hormone (TSH) as well as testosterone hormone in male adult rats. 
A total of 54 Sprague-Dawley albino adult male rats were divided into nine groups each of six rats, daily treated intraperitoneal with ZnO 
NPs two different doses (30 and 60) mg/kg in three different periods of time (7, 14, and 28) days, as following: Control groups (Groups 1, 
2, and 3): Respectively received intraperitoneal injection with distilled water for 7, 14, and 28 days, experimental groups (Groups  4, 
5, and 6): They were rats, respectively, received intraperitoneal dose (60 mg/kg) of ZnO NPs for (7, 14, and 28) days, and group (7, 8, 
and 9) experimental groups were rats, respectively, received intraperitoneal dose (30 mg/kg) of ZnO NPs for (7, 14, and 28) days. Data 
showed high significant decrease (P < 0.01) in level of T3, T4, TSH, and level of testosterone also decrease at high and low dose for 7, 14, 
and 28 days.

Keywords: T3, T4, testosterone, thyroid-stimulating hormone, zinc oxide nanoparticles

INTRODUCTION

Zinc oxide nanoparticles (ZnO NPs) are inorganic compounds, called multifunctional material due to unique chemical and physical properties.[1] Zinc is an 
essential metal it is an activator for more than 300 enzymes in 
the body.[2] ZnO NPs are one of most widely used in cosmetics 
and sunscreen because of their efficient UV absorption 
properties, they are used due to the antimicrobial properties 
in food packaging.[3] They are also being explored for their 
potential use as fungicides in agriculture.[4] As well ZnO 
nanoparticles are used in anticancer drugs and in biomedical 
applications.[5] Commonly considered to be a material with 
low toxicity because the ZnO is an essential trace element in 
human body and is existing in food or added as nutritional 
complement, so zinc attracts little care during assessment of 
toxicity of NPs.[6]

ZnO NPs can induce the formation of reactive oxygen 
species (ROS) that disrupt intracellular metabolic activities 
and the antioxidant system; these alterations permit generated 
ROS to interact with and damage DNA, lipids, carbohydrates, 
and proteins.[7] Thyroid gland is one of major endocrine glands 
of the body responsible of creating the hormones, thyroxin, 
and triiodothyronine which are essential for the proper 
organism development in particular for the nervous system 
and heart, normal growth, and skeletal maturation.[8] It is 
considered one of the main regulators of biological processes, 

during development, and childhood.[9] Decrease in the 
production of thyroid hormones means hypothyroidism.[10] 
Many evidence suggesting the role of zinc in the formation 
and function of thyroid hormones.[11] ZnO NPs cause higher 
levels of oxidative stress, resulting in inflammation, and cell 
toxicity.[12] Man testis has two main functions, the production 
of testosterone and the production of male germ cells, 
maintenance of spermatogenesis process needs testosterone, 
the androgen production is regulated by luteinizing hormone, 
while follicle-stimulating hormone is critical for initiation 
and maintenance of spermatogenesis.[13,14] Testosterone is the 
main androgenic hormone in males, it is largely produced 
by the Leydig cells of the testes, ZnO NPs were internalized 
by Sertoli cells and Leydig cells resulted in cytotoxicity in a 
time and dose-dependent manner through the induction 
of apoptosis, caused by increase in ROS related with loss of 

Corresponding Author: 
Noori M. Luaibi, Department of Biology, College of 
Sciences, University of Al-Mustansiriyah, Baghdad, Iraq. 
E-mail: sznl@uomustansiriyah.edu.iq

Received: Apr 25, 2019 
Accepted: Apr 28, 2019 
Published: Jan 20, 2020

DOI: 10.24086/cuesj.v4n1y2020.pp26-31

Copyright © 2020 Noori M. Luaibi, Noor A. Zayed. This is an open-access article 
distributed under the Creative Commons Attribution License.

Cihan University-Erbil Scientific Journal (CUESJ)

https://creativecommons.org/licenses/by-nc-nd/4.0/


Luaibi and Zayed: Zinc oxide nanoparticles effect on thyroid and testosterone hormones

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mitochondrial membrane potential, so injection of ZnO NPs 
produced structural alterations in the seminiferous epithelium 
and sperm abnormalities in male rats.[15]

MATERIALS AND METHODS

Adult Male Sprague-Dawley albino rats, age about 
2.5–3 months, average bodyweight 200–225 g. They were 
obtained from the National Center for Drug Control and 
Research/ministry of health, then transferred to the animal 
house of the college of science, Mustansyriah University. 
All animals were allowed to acclimatize to the laboratory 
conditions for 7 days before starting the study. They were 
kept in clean separated plastic cages with metal network cover 
under climate-controlled condition of the animal house with 
22–25 temperature, 60% humidity, 12 h light, and darkness 
period and allowed free access to food ad libitum and water. 
This study was conducted after obtaining the approval from the 
ethical consideration to deal with experimental animals that is 
the Ethical Committee of the College of Science, Mustansiriyah 
University and all ethical behavior with the laboratory animals 
was taken care and has priority in our work in accordance 
with the globally adopted guidelines of the animal care and 
experiments.

Preparation of ZnO NPs Solution

ZnO NPs used in this study was obtained from sky spring 
nanomaterials, they were in white to light yellow colored 
powder with 99.8% purity, particle size was 10–30 nm in 
diameter. The stock suspension was prepared by dissolving 1 g 
of powder ZnO in 10 ml of distilled water and then mixed by 
vortex for 10 min to prevent agglomeration, then distributed 
in to following groups:

 Group of 60 mg/kg of ZnO NPs (high dose) 120 µl of stock 
+880 µl of distal water.

 Group of 30 mg/kg of ZnO NPs (low dose) 60 µl of stock 
+940 µl of distal water.

Experimental Design

To study the effect of ZnO NPs, animals were divided into nine 
groups with six rats each as follows:
•	 Groups	1,	2,	and	3	(control	group),	respectively,	received	

intraperitoneal injection of distilled water for (7, 14, 
28) days.

•	 Groups	 4,	 5,	 and	 6	 (the	 experimental	 groups);	 rats,	
respectively, received intraperitoneal dose (60 mg/kg) of 
ZnO NPs for 7, 14, and 28 days.

•	 Groups	 7,	 8,	 and	 9:	 (The	 experimental	 groups);	 rats,	
respectively, received intraperitoneal dose (30 mg/kg) of 
ZnO NPs for 7, 14, and 28 days.

Measurement of the Levels of Hormones 
Concentration

It was represented by the enzyme immunoassay tests (TOSOH) 
for the quantitative determination of concentrations of thyroid 
gland hormones T3 according to Braverman et al.,[16] T4 
according to Ormston et al.,[17] and thyroid-stimulating hormone 
(TSH) according to Fisher.[18] Furthermore, the reproductive 
hormone testosterone was measured according to Sigberg.[19]

Collection of Blood Samples

The end of each experiment animals were completely 
anesthetized by diethyl ether for several minutes, and blood 
samples were obtained by heart puncture collected in to 
non-heparinized tubes used in biochemical examination, 
4 ml of blood collected from each rat was used to obtain 
sera (0.5–1.0) ml separated by centrifugation 3000 rpm for 
5	min,	then	they	were	kept	in	−20°C	until	analysis	for	the	
measurement of the level of TSH, measurement of the level of 
T3, measurement of the level of T4, and measurement of the 
level of testosterone.

RESULTS

Thyroid Hormone Function

Statistical analysis for the effect of ZnO NPs on T3, T4, and TSH 
serum levels is shown in Figures 1-3. T4 (µg/dl) serum level 
displayed high significant decrease (P < 0.01) in both treated 
groups (30, 60) mg/kg (3.10 ± 0.01), (2.90 ± 0.02) (µg dl), 
respectively, at day 7 compared to control groups (3.34 ± 
0.01) (µg/dl), at day 14 also showed high significant decrease 
(P < 0.01) at the same concentrations (2.71 ± 0.01), (2.21 ± 
0.02) (µg/dl) when compared with control group (3.35 ± 
0.01) (µg/dl), in addition to 28 period of time exposing to 
ZnO NPs observed high significant decrease (P < 0.01) of the 
level of T4 in different concentration (30, 60) mg/kg (1.91 ± 
0.02), (1.66 ± 0.01) (µg/dl) compared with control groups 
(3.36 ± 0.01) (µg/dl), as shown in Figure 1.

Statistical analysis of the level of T3 (ng/ml) showed 
high significant decrease (P < 0.01) in both treated groups 
(30, 60) mg/kg (0.685 ± 0.007), (0.600 ± 0.010) (ng/
ml), respectively, exposed to ZnO NPs for 7 days compared 
to the control groups (0.786 ± 0.008) (ng/ml), also there 
was high significant decrease (P < 0.01) in the level of T3 
at both concentrations (30, 60) mg/kg at 14 days (0.581 ± 
0.01), (0.438 ± 0.019) (ng/ml) in comparison to control 
groups (0.786 ± 0.008), and at 28 days (0.440 ± 0.01), 
(0.335 ± 0.01) (ng/ml) in comparison to control groups 
(0.795 ± 0.009) (ng/ml), as shown in Figure 2.

While serum level of TSH (µlU/ml) showed high significant 
decrease (P < 0.01) at different treatment durations exposing 
to ZnO NPs in both treated groups (30 and 60) mg/kg (0.186 ± 
0.007), (0.190 ± 0.010) (ng/ml), respectively, for 7 days 
compared to the control groups (175 ± 0.008) (ng/ml), also 
there was high significant decrease (P < 0.01) in the level of 
(TSH) at both concentrations (30 and 60) mg/kg in 14 days 
(0.188 ± 0.01), (0.195 ± 0.019) (ng/ml), respectively, in 
comparison to control group (0.175 ± 0.008), and at 28 days 
(0.191 ± 0.01), (0.198 ± 0.01) (ng ml) in comparison to 
control group (0.174 ± 0.009) (ng/ml), as shown in Figure 3.

Testosterone

Statistical analysis of the present study of ZnO NPs on testis 
function is observed in Figure 4 there was a high significant 
decrease (P < 0.01) in testosterone level at 30 and 60 mg/kg 
for 7 days (160.83 ± 1.16), (149.00 ± 1.29) Ng/dl, respectively, 
compared to control group (184.67 ± 1.05) Ng/dl. Testosterone 
level exhibited significant decrease (P < 0.01) at day14 



Luaibi and Zayed: Zinc oxide nanoparticles effect on thyroid and testosterone hormones

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when treated with ZnO NPs (30 and 60) mg/kg (138.50 ± 
0.99), (121.50 ± 0.99) Ng/dl when compared with control 
groups (181.00 ± 1.31) Ng/dl. There was significant decrease 
(P < 0.01) showed in testosterone level at a period of 28 days 
treatment (30 and 60) mg/kg (117.00 ± 1.52), (104.17 ± 1.42) 
Ng/dl in comparison to control groups (184.17 ± 1.24) Ng/dl.

DISCUSSION

Results of the present study are in agreement with previous 
study of ZnO NPs by Cheric and Rafieirad[20] showed that 
injecting ZnO NPs with three different doses (1.25, 2.5, and 5) 
mg/kg to 42 male rats divided into 7 groups, the blood samples 
of experimental with different doses were taken on the 1st, 3rd, 
4th, and 15th days after receiving Nano ZnO intraperitoneally 
(ip) to measure the amount of T3 and T4 hormone levels, 
results observed that acute injection of Nano ZnO reduced the 

amount of thyroid hormone T3 and T4, the effect occurred 
in different doses, but is more significant in the medium and 
long term, even in small amounts can cause negative effects 
on the activity of the thyroid gland and disrupt creation of 
thyroid hormones. Another study by Espanani et al.[21] who 
treated 48 male rats by ZnO NPs ip (5, 10, 20, and 40 mg/kg), 
after a 21 days period results showed significant increase in 
TSH hormone level in animals that were treated by high dose 
of ZnO NP, but those who treated by 5, 10, and 20 mg/kg 
do not show this effect. Dean et al.[22] Found a reduction in 
thyroid function T3, T4 when he fed chicken with Zn in diet 
at 73 ppm or 5280 ppm for 1–2 weeks, have demanded that 
high Zn intake change the production and secretion of thyroid 
hormones, they suggested that decrease in thyroid hormone 
function was due to reduced effects of thyroid gland might 
be induced by the reduced regulatory effect of pituitary gland 
on thyroid gland, and decreased circulating thyroid hormones 

Figure 2: Effect of different concentrations of zinc oxide nanoparticles 
(30 and 60) mg/kg on T3 levels of rats with different periods of time 
(7, 14, and 28) days in comparison with control groups and between 
treated	 groups	 themselves.	 **High	 significant	 decrease	 (≤0.01).		
A,B,CRepresents the significant difference between groups with days as 
a fixed factor and concentrations as a variable factor. a,b,cRepresents the 
significant difference between groups with concentrations as a fixed 
factor and days as a variable factor

Figure 1: Effect of different concentrations of ZnO NPs (30 and 60) 
mg/kg on T4 levels of rats with different periods of time (7, 14, and 28) 
days in comparison with control groups and between treated groups 
themselves.	**High	significant	decrease	(≤0.01).	A,B,CRepresents the 
significant difference between groups with days as a fixed factor 
and concentrations as a variable factor. a,b,cRepresents the significant 
difference between groups with concentrations as a fixed factor and 
days as a variable factor

Figure 3: Effect of different concentrations of zinc oxide nanoparticles 
(30 and 60) mg/kg on thyroid-stimulating hormone levels of rats 
with different periods of time (7, 14, and 28) days in comparison 
with control groups and between treated groups themselves. **High 
significant	decrease	(≤0.01).	A,B,CRepresents the significant difference 
between groups with days as a fixed factor and concentrations as a 
variable factor. a,b,cRepresents the significant difference between groups 
with concentrations as a fixed factor and days as a variable factor

Figure 4: Effect of different concentrations of zinc oxide nanoparticles 
(30 and 60) mg/kg on testosterone levels of rats with different 
periods of time (7, 14, and 28) days in comparison with control 
groups and between treated groups themselves. **High significant 
decrease	(≤0.01).	 A,B,CRepresents the significant difference between 
groups with days as a fixed factor and concentrations as a variable 
factor. a,b,cRepresents the significant difference between groups with 
concentrations as a fixed factor and days as a variable factor



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may be indicative of hypothyroidism due to Zinc toxicity. 
Lu et al.[23] Stated that Zn decrease the binding of T3 to its 
receptor in several preparations in vitro.

Kaya et al.[24] Conducted the experiment using 130 Hisex 
Brown laying hens from 56 weeks to 68 weeks of age, then 
they were divided into five zinc treatment groups (0, 25, 50, 
100, and 200) mg/zinc/kg diet respectively, the values of 100 
and 200 mg/Zn/kg decreased plasma level of T4 compared to 
control, while plasma level of T3 was reduced by 100 mg/Zn/
kg compared to groups fed less Zn, these data might suggest 
that a high intake of Zn changes production or secretion of the 
thyroid hormone.[25] Used 12 lambs and 12 goats that were 
divided into two equal groups as control and Zn groups in 
separate experiments, both species of animals in the Zn groups 
were fed a basal ration supplemented with zinc sulfate adjusted 
to 250 mg Zn/kg diet showed significant decrease in hormone 
level T3 and T4.[26] Randomly distributed 120 male rabbits into 
four groups, the control groups were fed on a basal diet with 
zinc-free premix, while experimental groups received the basal 
diet supplemented, Group 1 with 60 mg/kg nano ZnO/kg diet, 
Group 2 60/kg mg nano ZnO/kg diet, and Group 3 30/kg mg 
nano ZnO/kg diet, respectively, results observed that rabbits 
showed no significant changes among the treated groups in 
respect to serum TSH concentration. In contrast, Baweja et 
al.[27] demonstrated that the levels of TSH were raised after 
8 weeks of zinc supplementation in female Wistar rats. Another 
study that disagree with present report by Shirband et al.[28] 
that administered iron oxide NPs with three different dose 
(20 µg/kg, 50 µg/kg, and 150 µg/kg) for 15 days to male 
rats, results showed significant increase in T4 level in groups 
receiving a dose 50 µg/kg and caused significant decrease in 
TSH in the group receiving a dose 50 µg/kg and 150 µg/kg. It 
is likely that NP effects can be applied through the inhibition 
of endocrine pituitary axis hypothalamus which affects the 
hypothalamus, and probably due to decreased TSH levels. NPs 
affect hypothalamic pituitary thyroid axis and therefore affect 
the level of thyroid hormones that were demonstrated in this 
study. In a study by[29] used 32 adult male mice treated daily 
for 35 days: 5, 50, and 300 mg/kg ZnO NPs, respectively, while 
control group received distilled water orally for 35 consecutive 
days, results observed significantly changed in 50 and 300 
mg/kg ZnO NPs treated mice in epididymal sperm parameters 
including sperm number, motility, and percentage of 
abnormality, while significant decrease in seminiferous tubule 
diameter, seminiferous epithelium height, and maturation 
arrest was observed at 50 and 300 mg/kg ZnO NPs, this study 
established that ZNP has cytotoxic actions on testicular germ 
cells in a dose-dependent manner, NPs might also affect Sertoli 
cell functions.[30] Who treated 40 female Wistar rats with 
zirconium oxide NPs with a dose of 100, 200, and 400 ppm 
injected intraperitoneally showed a significant decrease in level 
of testosterone at high doses. Reports of a study by Mohammadi 
et al.[31]  injected Titanium dioxide TiO NPs intraperitoneally 
to Wistar rats weighing 150–250 g, 1 ml TiO2 NPs in doses 
(30 and 50) mg/kg, injection repeated every other day, results 
showed decrease in testosterone level, could be caused by 
adverse effects of NPs in the Leydig cells, as Leydig cells are 
testosterone production factories. Another study by Fuse et 
al.[32] showed that zinc NPs disorder leads to atrophy to the 
seminiferous tubules and impaired spermatogenesis in male 
rats. In a study by Shirvani et al.[33] was intraperitoneal injected 

of different doses (25, 50, and 100 mg/kg) of ZnO NPs (25 nm) 
to male Wistar rats showed decrease but no significant in level 
of testosterone, synthesis of testosterone is inhibited as a result 
of responding to the inflammation which is caused by ZnO NPs.
[34] Treated rats with ZnO NPs with dose (5, 10, 20, and 40 mg/
kg) results revealed significant increase in level of testosterone 
in blood serum of rats at high dose of ZnO NPs (40 mg/kg), 
results assured exposing to ZnO NPs damaged to public health 
and reduced fertility potential. ZnO NPs are related to (ROS), 
which results in an increase in DNA double-strand breakage and 
a decrease in sperm motility.[33] Kolesarova et al.[35]reported that 
metal NPs induce changes in reproductive organs, histology of 
laboratory animals and cause disruption in reproductive cells 
production and hormones. Thyroid hormone deficiency affects 
all tissues of the body including multiple endocrine changes 
that alter growth hormone, corticotrophin, gonadal function, 
and glucocorticoids, as primary hypothyroidism is associated 
with hypogonadotropic hypogonadism, so hypothyroidism 
decrease free testosterone concentration as thyroid hormone 
affects in sex hormone-binding globulin (SHBG).[36] Alterations 
in gonadal steroid genesis and pituitary functions have been 
stated in hypothyroid males, hypothyroidism was found to be 
associated with an increase in level of total cholesterol and 
reduction in the levels of testosterone and progesterone without 
any alteration in the levels of gonadotropins and estradiol, 
the decline in level of testosterone could be explained by 
reduction in serum triiodothyronine, a higher rate of alteration 
of testosterone to estradiol or the further decline in the rate of 
alteration of progesterone to testosterone.[37] Increasing in TSH 
level that was caused by ZnO NPs decreased gonadotropin-
releasing hormone secretion by negative feedback inhibition 
that led to LH and FSH reduction, while inhibin hormone that 
is usually released by Sertoli cells can affect FSH hormone 
level.[21] Considering the results of the above mentioned studies 
and the alterations in results from the present study, these 
outcomes could be related to the dosage of ZnO NPs used, 
animal species diversity, route of administration of NPs and 
different durations of exposing to NPs, NPs showed cytotoxic 
effect on testosterone and thyroid hormone levels in dose and 
time-dependent manner.

CONCLUSIONS

The results of this study showed that the size, doses, route 
of administration, and time depended can be a factor that 
effects thyroid hormone level and testosterone, the decrement 
in T3 andT4 can be caused by dose and duration of ZnO NPs, 
decrease in levels of thyroid hormones due to toxic effect of 
ZnO NPs that effects on the function of thyroid gland or on 
release of TSH. NPs affects the reproductive system of male 
by complex and varied mechanisms; results indicate that 
thyroid hormone deficiency has effects on testosterone level, 
as hypothyroidism decrease free testosterone concentration, 
also thyroid hormone has effects on SHBG.

ACKNOWLEDGMENTS

The authors would like to thank Mustansiriyah University 
(www.uomustansiriyah.edu.iq) Baghdad, Iraq, for its support 
in the present work.



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