Observation  |  Dermatol Pract Concept 2012;2(1):11 57

Human papillomavirus-induced periungual 
pigmented Bowen’s disease

Marigdalia K. Ramirez-Fort, M.D.1

1 Department of Surgery, University of Pittsburgh Medical Center, Pittsburgh, PA USA

Key words: human papillomavirus, pigmented Bowen’s disease, periungual Bowen’s disease

Citation: Case report: human papillomavirus induced periungual pigmented Bowen’s disease.  
Dermatol Pract Conc. 2012;2(1):11. http://dx.doi.org/10.5826/dpc.0201a11.

Editor: Alon Scope, M.D.

Received: September 23, 2011; Accepted: December 1, 2011; Published: January 31, 2012

Copyright: ©2012 Ramirez-Fort. This is an open-access article distributed under the terms of the Creative Commons Attribution License, 
which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Funding: None.

Competing interests: The authors have no conflicts of interest to disclose.

All authors have contributed significantly to this publication.

Corresponding author: Marigdalia K. Ramirez-Fort, M.D., Department of Surgery, University of Pittsburgh Medical Center,  
200 Lothrop St, Pittsburgh, PA15213. Tel. 908.285.8500. Email: ramirezfortmk@upmc.edu.

Case report

A 50-year-old male presented with an ill-defined hyperpig-

mented macule on the left pointer finger (Figure 1). The lesion 

was reportedly first evident one year prior to presentation. 

Clinically, the lesion was asymmetrical. Notably, there was 

central hypopigmentation and obliteration of acral architec-

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ture. Differential diagnosis included acral nevus, tinea nigra, 

subcorneal bleeding, exogenous pigmentation, melanoma 

and pigmented Bowen’s disease. 

Dermatoscopically, the lesion was chaotic with regard to 

the distribution of color (Figure 2). The lesion had different 

shades of brown and gray and its pattern was structureless. 

As noticed before by Cameron et al, a structureless pattern is 

Figure 1. Clinical view. [Copyright: ©2012 Ramirez-Fort.] Figure 2. Dermatoscopic view. [Copyright: ©2012 Ramirez-Fort.]



58 Observation  |  Dermatol Pract Concept 2012;2(1):11

Figure 3a–E. Dermatopathologic view. 

[Copyright: ©2012 Ramirez-Fort.]

A B

C D

E



Observation  |  Dermatol Pract Concept 2012;2(1):11 59

in keeping with the diagnosis of pigmented Bowen’s disease 

[1]. 

Histopathologic analysis of the lesion demonstrated a 

pigmented Bowen’s disease (Figure 3). There was acanthosis 

with focal hypergranulosis and parakeratosis. The epidermis 

was composed of atypical keratinocytes and a few scattered 

dyskeratotic cells. The superficial dermis had a light peri-

vascular lymphocytic infiltrate. Polymerase chain reaction 

sampling was positive for human papillomavirus (HPV), fur-

ther confirming the diagnosis of an HPV-induced pigmented 

Bowen’s disease. The lesion was excised completely and the 

surgical defect was closed primarily.

Discussion 

Bowen’s disease may be induced by chronic exposure to 

forms of electromagnetic radiation, such as ultraviolet light 

(UV) or x-ray. Bowen’s disease may also be induced by infec-

tion with HPV, as in the case presented above. Bowen’s dis-

ease induced by HPV usually occurs on genital skin; multiple 

regional lesions are termed “bowenoid papulosis.” 

Bowen’s disease is further categorized into pigmented or 

nonpigmented. it has been speculated that the source of pig-

ment in Bowen’s disease may represent a collision between a 

solar lentigo and Bowen’s disease. Although this explanation 

is reasonable for pigmented Bowen’s disease on cutaneous 

sites chronically exposed to sunlight, it does not explain the 

pigmentation of “bowenoid papulosis” on genital skin or the 

case presented above. A more probable hypothesis considers 

neoplastic pigmentation as a direct reflection of the pigmen-

tation present in the initial keratinocytes to proliferate. The 

hypothesis holds validity in the setting of a benign seborrheic 

keratosis or malignant lesion of Bowen’s disease.

Some patients with bowenoid papulosis on genital skin 

develop subsequent periungual Bowen’s disease. Reported 

cases have found the same HPV strains in genital and peri-

ungual lesions, suggesting anogenital-digital spread of HPV 

as a possible etiology for HPV-positive periungual Bowen’s 

disease [2]. The literature reports only one other case of a 

pigmented, periungual Bowen’s disease by Hu et al [3]. 

Although the HPV status of the lesion is unknown, the group 

clearly demonstrated the utility of dermatoscopy in evalua-

tion and further monitoring of disease progression.

References

1. Cameron A, Rosendahl C, Tschandl P, Kittler H. Dermatos-

copy of pigmented Bowen’s disease. J Am Acad Dermatol. 

2010;62(4):597–604.

2. Hu SC, Chiu HH, Chen GS, et al. Dermoscopy as a diagnostic and 

follow-up tool for pigmented Bowen‘s disease on acral region. 

Dermatol Surg. 2008;34(9):1248–53.

3. Shim WH, Park HJ, Kim HS, et al. Bowenoid papulosis of the 

vulva and subsequent periungual Bowen‘s disease induced by the 

same mucosal HPVs. Ann Dermatol. 2011;23(4):493–6.