Dermatology: Practical and Conceptual


Research Letter | Dermatol Pract Concept. 2022;12(4):e2022206 1

Dermatologic Manifestations of 
Thymoma-associated Multiorgan Autoimmunity 

(TAMA) Syndrome: Cutaneous Signs of an  
Immune Dysregulation

Luigi Rossiello1, Amalia Lupoli2, Giuseppe Cicala1, Gianfranco De Dominicis3,  
Vittorio Tancredi2, Stefano Caccavale2

1 Department of Dermatology, A.O.R.N. “A. Cardarelli”, Naples, Italy.

2 Department of Dermatology, “Luigi Vanvitelli” University of Naples, Italy.

3 Department of Pathology, A.O.R.N. “A. Cardarelli”, Naples, Italy.

Key words: TAMA syndrome, myastenia gravis, GVHD, thymoma

Citation: Rossiello L, Lupoli A, Cicala G, De Dominicis G, Tancredi V, Caccavale S. Dermatologic manifestations of thymoma-associated 
multiorgan autoimmunity (TAMA) syndrome: cutaneous signs of an immune dysregulation. Dermatol Pract Concept. 
2022;12(4):e2022206. DOI: https://doi.org/10.5826/dpc.1204a206

Accepted: February 25, 2022; Published: October 2022

Copyright: ©2022 Rossiello et al. This is an open-access article distributed under the terms of the Creative Commons  
Attribution-NonCommercial License (BY-NC-4.0), https://creativecommons.org/licenses/by-nc/4.0/, which permits unrestricted 
noncommercial use, distribution, and reproduction in any medium, provided the original authors and source are credited.

Funding: None.

Competing interests: None.

Authorship: All authors have contributed significantly to this publication.

Corresponding author: Vittorio Tancredi, MD, Dermatology Unit, Department of Mental and Physical Health and Preventive Medicine, 
University of Campania Luigi Vanvitelli, Via Sergio Pansini, 5, 80131 Naples, Italy; phone: +39 3313919797; fax: +39 0815468759; 
E-mail: tancredivittorio@ymail.com

Introduction

Thymoma-associated multiorgan autoimmunity (TAMA) 

syndrome is the consequence of auto-reactive T-cells activa-

tion developing in the setting of a thymoma, which mediates 

graft-versus-host-disease (GHVD)-like reactions in several 

tissues, including skin [1].

Case Presentation

A 61-year-old man was admitted in our Dermatology 

 Department for a skin rash appearing concomitantly to a 

nodal relapse of a malignant thymoma. Cutaneous examina-

tion revealed a confluent erythematous and papulo-squamous 

eruption involving most of his face, trunk, bilateral upper 

and lower extremities. His palms and soles presented conflu-

ent pink tender papules. Moreover, multiple chronic painful 

erosions affected the oral mucosa (Figure 1). The patient re-

ported general malaise with deep asthenia and an abundant 

chronic diarrhea.

A skin biopsy was performed and histologic examina-

tion revealed an interface and perivascular dermatitis in 

the dermis. Epidermis showed psoriasiform hyperplasia, 

diffuse parakeratosis and spongiosis, hypogranulosis, ne-

crotic keratinocytes with intense eosinophilic cytoplasm 

(Figure 2). Although several differential diagnoses (includ-

ing drug reaction, viral exanthema, pityriasis lichenoid, and 

sub-erytrodermic psoriasis) were considered, relying on 



2 Research Letter | Dermatol Pract Concept. 2022;12(4):e2022206

Figure 1. Erythematous and scaly papulosquamous eruption.

Figure 2. (A) Histology showing epidermal hyperplasia (H&E, 40X). Higher magnification highlights parakeratosis, epidermal spongiosi , 

hypogranulosis and some apoptotic keratinocytes characterized by intensely eosinophilic cytoplasm. (B) The underlying dermis contains few 

infiltrates of lymphocytes (H&E, 200X).

anamnesis, histology and literature, a GVHD-like reaction 

occurring in the setting of a malignant thymoma was finally 

diagnosed.

In addition, our patient developed a progressive muscle 

weakness. A thymoma-related myasthenia gravis was diag-

nosed after the detection of autoantibodies directed against 



Research Letter | Dermatol Pract Concept. 2022;12(4):e2022206 3

acetylcholine receptor and electrophysiological evaluation of 

neuromuscular junctions.

After three months our patient died as a result of the 

rapidly progressive clinical deterioration and the hypoxemic 

respiratory failure consequent to a lung infection and myas-

thenia gravis.

Myasthenia gravis is the most typical paraneoplastic syn-

drome associated with thymoma; it does not always develop 

at diagnosis, but it has a high impact of morbidity and mor-

tality [2].

GVHD-like reactions are rare immune response that 

occur particularly at level of skin, intestine, or liver, which 

resemble GVHD on histopathology, except for graft lympho-

cytes [3]. The diagnosis requires the exclusion of the main 

causes of a real GHVD, such as hematopoietic stem cell 

transplantation (HSCT) and transfusion of non-irradiated 

blood. Being not associated to HSCT, Waldhera et al decided 

to collect the GVHD-like reactions occurred in the setting of 

a thymoma under the umbrella name of TAMA syndrome 

[1]. First described cases were characterized by a constant 

colon involvement; subsequently, TAMA reactions were de-

tected in multiple organs, including not only gastrointestinal 

tract, but also skin, thyroid and liver [4].

Conclusions

We remark the uniqueness and interest of this case, since 

TAMA syndrome is a very rare disorder and with few cases 

reported in literature to date.  The dermatologic manifesta-

tions of TAMA syndrome consist of diffuse papulo-squamous 

rash, often involving palms and soles, with possible lesions 

of oral mucosa. TAMA syndrome should be always kept in 

mind on a thymoma background when these specific skin 

signs are associated with systemic manifestations, first of all 

diarrhea. 

References

1. Waldhera A, Maverakis E, Mitisiades N, Lara PN,  Fung 

MA, Lynch PJ. Thymoma associated multiorgan autoimmunity: 

a graft-versus-host-like-disease. J Am Acad Dematol.  2007; 

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17433850. 

2. Tian W, Li X, Sun Y, Wang J, Jiang G, Tong H. Myasthenia gravis 

affects overall survival in patients with thymoma: an analysis of 

multicentre database using propensity score matching. Interact 

Cardiovasc Thorac Surg. 2021;33(2):250-257. DOI: 10.1093/

icvts/ivab074. PMID: 34151968. PMCID: PMC8691723.

3. Holder J, North J, Bourke J, Colloby P, Fletcher A, Graham-Brown 

R, Whaley K. Thymoma-associated cutaneous graft-versus-host-

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4. Warren S, Nehal K, Querfeld C, Wong R, Huang J, Pulitzer M. 

Graft-versus-host disease-like erythroderma: a manifestation 

of thymoma-associated multiorgan autoimmunity. J Cutan 

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26509934. PMCID: PMC5072282.