Dermatology: Practical and Conceptual


Review  |  Dermatol Pract Concept 2019;9(2):1 75

Dermatology Practical & Conceptual

Nonsurgical Options for the Treatment 
of Basal Cell Carcinoma

John Paoli1,2, Johan Dahlén Gyllencreutz3, Julia Fougelberg1,2, Eva Johansson Backman1,2, 
Maja Modin1,2, Sam Polesie1,2, Oscar Zaar1,2

1 Department of Dermatology and Venereology, Institute of Clinical Sciences, Sahlgrenska Academy, University of Gothenburg, Gothenburg, 

Sweden

2 Region Västra Götaland, Sahlgrenska University Hospital, Department of Dermatology and Venereology, Gothenburg, Sweden

3 Department of Dermatology and Venereology, Skaraborg Hospital, Skövde, Sweden

Key words: basal cell carcinoma, destructive therapy, topical drugs, radiotherapy, hedgehog inhibitors

Citation: Paoli J, Dahlén Gyllencreutz J, Fougelberg J, Johansson Backman E, Modin M, Polesie S, Zaar O. Nonsurgical options for the 
treatment of basal cell carcinoma. Dermatol Pract Concept. 2019;9(2):75-81. DOI: https://doi.org/10.5826/dpc.0902a01

Accepted: November 15, 2018; Published: April 30, 2019

Copyright: ©2019 Paoli et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, 
which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Funding: None.

Competing interests: The authors have no conflicts of interest to disclose.

Authorship: All authors have contributed significantly to this publication.

Corresponding author: John Paoli, MD, Assoc. Prof, Department of Dermatology and Venereology, Sahlgrenska University Hospital, 413 
45 Gothenburg, Sweden. Email: John.paoli@vgregion.se

Objectives: The aim of this review article is to summarize the effectiveness, potential adverse events, 
and indications of the main nonsurgical treatment alternatives for basal cell carcinoma.

Methods: An extensive literature review was carried out. The most relevant articles were discussed 
and selected by the authors in order to provide a brief but evidence-based overview of the most com-
mon nonsurgical methods used for treating basal cell carcinoma.

Results: Although surgery and Mohs micrographic surgery are often considered the optimal treatment 
options for basal cell carcinoma, these tumors can also be treated successfully with destructive tech-
niques (eg, curettage alone, cryosurgery, or electrodesiccation), photodynamic therapy, topical drugs 
(eg, 5-fluorouracil, imiquimod, or ingenol mebutate), radiotherapy, or hedgehog pathway inhibitors. 
When choosing between these alternatives, physicians must take into consideration the tumor’s size, 
location, and histopathological subtype. Special care should be taken when treating recurrent tumors. 
Furthermore, physician experience is of great importance when using destructive techniques. Finally, 
patient preference, potential adverse events, and cosmetic outcome should also be considered.

Conclusions: Dermatologists and physicians treating basal cell carcinoma should have knowledge of 
and experience with the large arsenal of therapeutic alternatives available for the successful, safe, and 
individualized management of patients with basal cell carcinoma.

ABSTRACT

Introduction

Basal cell carcinoma (BCC) is by far the most common 

cancer type in humans [1]. In Europe, reports show BCC 

incidence rates between 77 and 158 per 100,000 person-years 

age-standardized to the European standard population [1]. 

Exposure from ultraviolet radiation is believed to be the main 

risk factor for developing BCCs. This belief is supported by 

the fact that the majority of BCCs are found on sun-exposed 

body parts in middle-aged to elderly people [1,2]. The exact 



76 Review  |  Dermatol Pract Concept 2019;9(2):1

inoperable BCCs may benefit from radiotherapy or hedgehog 

pathway inhibitors (HPIs) [1,16,17].

The aim of this review is to summarize the effectiveness, 

potential adverse events, and indications of the main nonsur-

gical treatment alternatives for BCC.

Destructive Methods

Curettage Alone

BCCs amenable to treatment with curettage alone are pri-

mary tumors with a superficial or nodular growth pattern 

with well-defined borders not involving the free margin of 

the eyelid, mucosal lip, or subcutaneous fat. For nBCCs, 

the size is recommended to be <6 mm in high-risk areas, 

and below 20 mm elsewhere. The few studies on curettage 

alone were carried out many years ago and did not use a 

unifying technique. In some countries, including the USA, a 

shave biopsy is performed first, removing the tumor almost 

completely prior to curettage. In other countries, including 

Sweden, curettage is performed directly, removing the entire 

tumor with the curette. Nevertheless, 3 retrospective studies 

have reported similar clearance rates of 89.9%-96.0% after 

5 years of follow-up. Better cosmesis with minimal hypopig-

mentation or scarring was observed in patients treated with 

curettage alone compared with those treated with curettage 

and electrodesiccation (C&ED) [18-20].

Curettage and Electrodesiccation

Although C&ED has been used for decades as a simple and 

easily performed therapy for low-risk BCCs, there is a lack 

of randomized controlled studies on the method. Several ret-

rospective studies show its effectiveness for correctly selected 

lesions (93.0%-96.9% clearance rates after 5 years of follow-

up), but many studies fail to provide precise descriptions on 

the materials and technical protocols that are used [21,22]. 

In the latest American Academy of Dermatology guidelines, 

C&ED is one of the recommended treatment options for 

carefully selected low-risk primary lesions [16]. In terminal 

hair-bearing skin, with a potential follicular extension of 

the tumor (scalp, pubic, and axillary regions as well as the 

beard area in men), C&ED is considered less effective [17]. 

The effectiveness depends heavily on the physician’s skills 

and technique, and therefore proper training is important 

[23,24]. Furthermore, the cosmetic outcome is regarded as 

inferior compared with standard excision and therefore it is 

best avoided in cosmetically sensitive areas [25].

Cryotherapy

Cryotherapy with liquid nitrogen (−196.5°C) has been used to 

treat nonmelanoma skin cancer since the early 1960s [26]. It 

is a well-established treatment method for small, well-defined 

origin of BCC is not established, but they are considered to 

arise from keratinocyte cells located in the basal layer and in 

the dermo-epidermal junction zone [3].

The World Health Organization classification of BCC 

includes superficial, nodular, micronodular, and infiltrating 

basal cell carcinoma. Moreover, the group includes fibroepi-

thelial basal cell carcinoma with adnexal differentiation, baso-

squamous carcinoma, and keratotic basal cell carcinoma [4].

The infiltrative, morpheaform, and micronodular sub-

types grow in a more aggressive way, whereas superficial and 

nodular subtypes generally have a less aggressive course and 

generally respond better to treatment [5].

Clinically, dermoscopy can assist physicians in the correct 

diagnosis of BCC, including the histopathological subtype, 

with relatively high diagnostic accuracy [6-8]. The first 

articles on BCC focused on pigmented BCCs showing a lack 

of pigment network in combination with blue-gray ovoid 

nests, multiple blue-gray dots/globules, leaf-like areas, spoke 

wheel/concentric areas, ulceration, and/or arborizing vessels 

[6,7,9]. In nodular BCC (nBCC), arborizing vessels are a com-

mon finding, sometimes together with ulceration [10]. Under 

polarized light, shiny white lines can also be found [11]. 

Superficial BCCs (sBCCs) characteristically show fine, short 

telangiectasias on a whitish-red background and multiple 

erosions [7,8,12]. The dermoscopic findings in more aggres-

sive forms of BCC are less well studied. In infiltrative BCCs, 

arborizing vessels are usually thinner than in nBCCs. Other 

findings include white-red structureless areas and ulceration. 

The morpheaform variant is often feature-poor, even when 

using dermoscopy. A white background can often be seen, 

sometimes together with a smaller number of thin arborizing 

vessels [13].

During the latest decades, insights into genetic and molec-

ular changes behind the origin of BCC have increased. These 

understandings have to some extent been derived from study-

ing patients with different genetic syndromes predisposing 

them to BCC development (eg, Gorlin syndrome) who have a 

higher risk of developing BCCs [14]. Individuals with Gorlin 

syndrome develop multiple BCCs starting at an early age and 

the responsible mutation lies in the Hedgehog (Hh) receptor 

Patched 1 (PTCH1) gene that mediates Sonic Hh signaling. 

Research has reported abnormal function and mutations in 

PTCH1 in up to 90% of BCCs, making it a target for drug 

development [3].

With regard to the management of BCC, surgery (includ-

ing Mohs micrographic surgery) is considered the gold stan-

dard [1,15,16]. Nevertheless, with the rising number of 

patients with BCC, increasing health care costs, and the lack 

of access to dermatologists in many countries, nonsurgical 

options may be considered. Many low-risk tumors can be suc-

cessfully managed with destructive methods, photodynamic 

therapy (PDT), or topical medications, while advanced or 



Review  |  Dermatol Pract Concept 2019;9(2):1 77

[37,44], while the photosensitizing prodrug used does not 

seem to influence the outcome [45,46]. The histopathologi-

cal subtype influences the result of PDT as pigmented, mor-

phoeic, and infiltrative BCCs are more resistant to treatment. 

The clearance rates following PDT for sBCCs range from 

76% to 82% in several studies [37,38,42], but could be as 

low as 58% [47]. The success rate is slightly lower for nBCCs 

in general (53%-76%) [36,39-41]. Debulking nBCCs with 

curettage prior to PDT provides long-term clearance rates of 

62%-73% [36,41,43] compared with light curettage without 

debulking, which provides more varying clearance rates of 

33%-76% [38,40]. Fewer recurrences have been observed 

when patients with nonaggressive BCCs receive PDT in 2 

sessions (91%) compared with only 1 session of PDT (68%) 

with a follow-up of 6 years [48]. Three randomized controlled 

trials have compared the efficacy of aminolevulinic acid PDT 

and methyl aminolevulinate (MAL) PDT for BCCs, showing 

comparable effectiveness [45,46,49].

Modifications to PDT have been tested to decrease the 

recurrence rate for BCC. Daylight PDT may provide similar 

clearance rates compared with conventional PDT, but with 

less pain [50]. However, larger studies with longer follow-up 

times are needed to confirm its effectiveness. Treating BCCs 

with a fractional laser prior to PDT may improve the depth 

of penetration of the prodrug and has shown lower recur-

rence rates than conventional PDT for high-risk BCCs (19% 

vs 44%) [51]. 

A meta-analysis by Wang et al showed no significant dif-

ferences in recurrence rates when comparing PDT to either 

cryotherapy or pharmacological treatment. However, sur-

gery proved to have significantly lower risk of failure when 

compared with PDT. The same analysis showed a “good to 

excellent” cosmetic result comparing PDT to surgery or cryo-

therapy, but no significant differences when comparing it to 

other medical treatments [52].

Topical Medication

5-Fluorouracil

Topical 5-fluorouracil (5-FU) 5% inhibits DNA synthesis and 

was the first topical therapy approved for the treatment of 

sBCC [53]. However, compared with PDT and imiquimod, 

fewer well-designed studies evaluating this indication have 

been published [47,54]. In a prospective, single-arm, clini-

cal study, 29 patients with 31 histopathologically confirmed 

sBCCs were treated with 5-FU applied twice daily for up 

to 12 weeks. The lesions were excised 3 weeks after treat-

ment with a histopathological clearance rate of 90% [53]. 

A single-blind, noninferiority, randomized controlled trial 

with 601 histopathologically confirmed sBCCs compared 

the use of PDT (2 sessions with an interval of 1 week) with 

topical imiquimod (once daily, 5 times/week for 6 weeks) and 

primary BCCs without sclerosing or infiltrative growth pat-

terns. Location below the knee is a relative contraindication 

because of prolonged wound healing [27]. Large treatment 

series have shown clearance rates of 97%-99% after at least 

5 years of follow-up [28,29]. As for C&ED, there are few 

prospective randomized trials on cryotherapy for BCCs and 

different techniques and protocols are used, making com-

parisons difficult. Most studies on cryotherapy involve prior 

curettage. Curettage is regarded as a help in delineating the 

lateral extension and depth of the tumor and also to diminish 

the amount of tumor mass that has to be sloughed off during 

the healing process following cryotherapy.

Three prospective studies using a standardized curettage 

and cryotherapy protocol have reported very high clearance 

rates of >98% for BCCs located in the face and scalp area, 

especially on the nose and ear [29-31]. The protocol in all 3 

studies involves curettage followed by a double freeze-thaw 

cycle. Thorough curettage with different sized curettes is first 

used to debulk the tumor and to identify the lateral and deep 

extension. Following hemostasis with a 50% iron chloride 

solution, a 3-mm thick, open and appropriately sized neo-

prene cone is placed around the ulcerated area. Subsequently, 

freezing takes place with maximal continuous liquid nitrogen 

spray using a hand-held Cry-Ac (Brymill Corp., Ellington, 

CT, USA) through a size B nozzle, held horizontally and at 

a distance of approximately 1 cm while moved in circular 

motions over the treatment area. Freezing is interrupted when 

the lateral spread of freeze (halo) has reached a minimum 

of 5 mm outside the tumor border or 1.0-1.5 mm outside 

the neoprene cone. The thaw time of the halo should last at 

least 60 seconds. After complete thawing, the freeze-thaw 

cycle is repeated.

Cryotherapy in a double freeze-thaw cycle as described 

above but without previous curettage has also shown excel-

lent clearance rates for well-defined BCCs of the eyelids with 

a 10-year recurrence rate of <0.5% [32].

Photodynamic Therapy

PDT is an alternative approach to treat superficial nonmela-

noma skin cancer, including BCC [33]. PDT for BCC is often 

well tolerated, but pain and/or a burning sensation is often 

experienced during the illumination phase [34]. After PDT, 

localized erythema and edema are common, followed by the 

formation of erosions and crusts with healing over a period 

of 2-6 weeks [1]. PDT is considered an interesting alternative 

treatment because it is noninvasive, it targets neoplastic cells 

selectively, it is cost-effective, and it offers a good cosmetic 

outcome [35,36].

There are many different treatment protocols for PDT 

and results vary depending on the BCC subtype [36-42], prior 

curettage [36,38,40,41,43], and number of PDT sessions 



78 Review  |  Dermatol Pract Concept 2019;9(2):1

treatment of actinic keratosis through induction of primary 

necrosis of tumor cells, and neutrophil-mediated, antibody-

dependent cellular cytotoxicity of residual malignant cells 

[60]. IM comes in 2 concentrations: 0.015% for the face and 

0.05% for the trunk. Recently, several case reports of success-

ful off-label treatment of sBCC with topical IM have been 

reported [61-65]. In a retrospective study, including 7 patients 

with sBCC treated with IM gel 0.05% once daily for 2-7 days, 

there were no clinical signs of recurrence after 2-14 months 

[66]. A phase IIa study including 60 patients with safety as 

the primary endpoint evaluated the treatment of sBCC with 

IM at concentrations of 0.01/0.025/0.05% applied for 2 

consecutive days or on day 1 and 8. The incidence of adverse 

events was generally low. The efficacy (secondary endpoint) 

appeared to be dose-related with up to 63% histopatho-

logical clearance with the highest dosage after 12 weeks [60]. 

Another phase I/II clinical study including 28 BCCs (6 nBCCs 

and 22 sBCCs), which were unresponsive to earlier treatment 

or unsuitable for surgery, treated with sap from E peplus 

once daily for 3 days, showed complete clinical clearance in 

82% after 1 month and 57% after 2-31 months, while sBCCs 

smaller than 16 mm (n = 9) had a 78% clearance after a 

mean follow-up of 15 months [67]. For sBCCs unsuitable for 

conventional treatment, IM can offer an alternative treatment 

strategy in selected patients, but close follow-up is advised.

Radiotherapy

The vast majority of BCCs are treated with surgery or the 

destructive methods listed above. Although rare, some BCCs 

are neglected and/or are inaccessible to standard treatments. 

In these rare cases, radiotherapy (RT) may be considered. 

Patients unwilling to undergo surgery or with significant 

comorbidities might also be candidates for this treatment 

option. There are 3 different radiation modalities: low-energy 

X-rays, brachytherapy, and high-energy RT. Treatment suc-

cess is generally 90%, which is in line with surgical treatment 

[68]. However, only a few clinical trials have been conducted, 

and there is no consensus on optimal treatment duration and 

which radiation modality should be selected [69]. RT is given 

in fractions of 30-70 Gy over 2-7 weeks, which requires mul-

tiple visits to a specialized clinic. A prospective randomized 

trial selecting patients for surgery or RT with a subrandom-

ization to long and short RT would be helpful to clarify which 

radiation modality is superior with regard to recurrence rates. 

Due to long-term consequences such as radiodermatitis and 

the risk of secondary malignancies, mainly patients over 60 

years should be considered for RT. In a Canadian study, the 

cost of managing high-risk facial BCCs with RT was higher 

than that of Mohs micrographic surgery [70]. Moreover, prior 

to initiation, a multidisciplinary decision with an experienced 

radiation oncologist and thorough patient information is 

topical 5-FU (twice daily for 4 weeks). In this study, topical 

5-FU was noninferior to PDT and imiquimod was superior to 

PDT. Furthermore, topical 5-FU and imiquimod were more 

often associated with mild to moderate adverse events in the 

treatment area than was PDT [55]. A 5-year follow-up on the 

study recently declared the probability of tumor-free survival 

to be 62.7% for PDT, 80.5% for imiquimod, and 70.0% for 

5-FU [56]. In summary, 5-FU is approved for treatment of 

sBCC, appears to be noninferior to PDT, and is generally well 

tolerated. Therefore, it can be considered a therapeutic option 

in the treatment of sBCC.

Imiquimod

Imiquimod works as an immunomodulator by activating 

Toll-like receptor 7, resulting in stimulation of dendritic cells 

residing in both the epidermis and dermis to attract natural 

killer cells and produce cytokines via proliferation of B lym-

phocytes. The treatment regimen is classically topical imiqui-

mod 5% applied once daily, 5 days per week for 6 weeks in 

sBCC and 12 weeks in nBCC.

Jansen et al recently presented 1-, 3-, and 5-year efficacy 

data on imiquimod in more than 100 histopathologically 

confirmed sBCCs. After 1 year, 83.5% of the sBCCs were 

successfully treated and at 5 years 80.5% still showed no 

recurrences [56]. Similar findings were made by Bath-Hextall 

et al, who treated 254 BCC lesions (both superficial and 

nodular) with imiquimod with clearance rates of 85.1% for 

sBCCs and 81.8% for nBCCs at 3 years [57]. Williams et al 

later presented 5-year results from the same cohort with an 

83.8% success rate for sBCC and 81.1% for nBCCs [58]. In 

these 2 studies, the recurrences were based on clinical assess-

ment and were not histopathologically confirmed.

With regard to aesthetic outcome, Arits et al reported 

in a large randomized controlled trial that imiquimod and 

MAL-PDT have similar results with good to excellent out-

comes in 61.4% and 62.4% of the cases, respectively [55]. A 

cost-effectiveness study on topical treatments for BCC show 

that imiquimod and 5-FU are superior to MAL-PDT at 12 

months but longer follow-up time is needed to assess the cost-

effectiveness between imiquimod and 5-FU [59].

When compared with MAL-PDT for sBCCs, imiquimod 

is known to be superior in general but a subgroup analysis 

performed by Roozeboom et al in a randomized controlled 

trial with 200 lesions in each treatment arm revealed that 

PDT might outperform imiquimod. Also, in patients over 60 

years of age with sBCC on the lower extremities, MAL-PDT 

was significantly more effective than imiquimod at 1-year 

follow-up [47].

Ingenol Mebutate

Ingenol mebutate (IM) gel is a topical agent derived from 

sap from the plant Euphorbia peplus, which is approved for 



Review  |  Dermatol Pract Concept 2019;9(2):1 79

ence with the techniques; the expected adverse events; patient 

preference; and cosmetic outcome.

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mandatory. Finally, recurring BCC after RT is a surgical chal-

lenge and this should be carefully considered.

Hedgehog Pathway Inhibitors

HPIs have emerged as an important treatment option for 

locally advanced BCC or in the rare event of metastatic BCC 

[71]. There are 2 commercially available HPIs: vismodegib 

and sonidegib. In a systematic review investigating vismo-

degib, 65% of patients with locally advanced BCC expe-

rienced complete or partial response, whereas 31% had a 

complete response rate. The corresponding numbers for meta-

static BCC were 34% and 4%, respectively. However, among 

803 patients, 31% discontinued the treatment because of side 

effects [72]. Muscle spasms, dysgeusia, fatigue, weight loss, 

and alopecia are common. In addition, treatment with HPI 

is associated with high costs (approximately €4500/month).

Neoadjuvant treatment with HPIs prior to surgery for 

selected cases has been evaluated with positive results [73], 

but prospective randomized controlled trials are lacking. 

When selecting this treatment option there is risk for mul-

tifocal remaining disease, which might complicate surgery. 

Although there is no consensus, concurrent treatment with 

RT and HPIs may be evaluated in selected cases [74].

Conclusions

This review shows that several effective nonsurgical therapeu-

tic alternatives exist for the successful, safe, and individual-

ized management of BCC. Physicians managing patients 

with BCCs need to be aware of the indications, effectiveness, 

and potential adverse events of this wide range of treatment 

methods so that they are able to select the most adequate 

method for each patient and clinical scenario. Physicians also 

need specific training in each method in order to reproduce 

the same clinical outcome seen in the aforementioned stud-

ies. Furthermore, physicians should be aware of the lack of 

randomized controlled trials for many of the nonsurgical 

procedures mentioned, even though they are regularly carried 

out by the dermatological community.

More extensive guideline documents for the general 

management of BCCs have been published by associations 

such as the European Dermatology Forum [1], the American 

Academy of Dermatology [16], and the National Comprehen-

sive Cancer Network [17], and this review does not intend to 

substitute any of them. However, the techniques mentioned 

herein can provide excellent results for the ever-growing num-

ber of patients who can benefit from nonsurgical approaches 

to managing BCC as long as the treating physician takes into 

account tumor size, location, and histopathological subtype; 

whether the tumor is primary or recurrent; their own experi-



80 Review  |  Dermatol Pract Concept 2019;9(2):1

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