id	author	title	date	pages	extension	mime	words	sentence	flesch	summary	cache	txt
easat-267	RS, Balgir 	Protective Resistance by Human G6PD Enzyme Deficiency and Hemoglobin Variants Against Malaria and Natural Selection: Further Evidence from Review of New Studies	2019	9	.pdf	application/pdf	8179	342	46	Malaria parasites have co-evolved with the host and constitute an important deriving evolutionary force behind common erythrocyte variants such as thalassemia, sickle cell disease, Hb C, Hb D, Hb E and G6PD deficiency and other erythrocyte anomalies. This trend for an inverse relationship between sickle cell disorders and G6PD deficiency due to disequilibrium in major scheduled caste and tribal communities of Central-Eastern India, is fascinating one [1,2].	cache/easat-267.pdf	txt/easat-267.txt