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Nova Biotechnologica 9-2 (2009)                                                                               107 
 

 
 

Cr AND Ni SIMULTANEOUS PHYTOTOXICITY AND 
MUTAGENICITY ASSAY  

 
AGÁTA FARGAŠOVÁ, JANA LIŠTIAKOVÁ 

 
Department of Ecosozology and Physiotactics, Faculty of Natural Sciences, Comenius 

University in Bratislava, Mlynská dolina, SK-842 15 Bratislava, Slovak Republic 
(fargasova@fns.uniba.sk) 

 
Abstract: For genotoxicity study simultaneous phytotoxicity and mutagenicity assay with Vicia sativa L. 
var. Klára was used. For phytotoxicity the following rank orders of growth inhibition can be arranged: for 
roots: Ni(II) > Cr(VI) > Cr(III); for shoots: Ni(II) > Cr(VI) ≥ Cr (III). For mutagenicity assay root tips of V. 
sativa were used and chromosome aberrations were determined at least in 500-anatelophases. All tested 
metals exerted in V. sativa a significant increase of chromosomal aberration rate in applied concentrations. 
Maximum of aberrations invoked Cr(VI) and the rank order of aberrations fall was: Cr(VI) > Ni(II) > 
Cr(III). Genotoxic effects of metals were  determined by analysis of micronuclei frequency in the pollen  
tetrads of Tradescantia plants. None of tested metal significantly stimulated micronuclei frequency and 
genotoxic effect was decreased in order: Cr(VI) ≥ Ni(II) > Cr(III). 
 
Key words: chromium, nickel, Vicia sativa, phytotoxicity, chromosomal aberation assay, Tradescantia 
micronucleus assay  

 
1. Introduction 

 
Vascular plants have been found to be highly effective for recognizing and 

predicting metal stress in the environment (growth inhibition, reduction of biomass 
production, changes in water absorption and translocation) (SHANKER et al., 2005; 
SZÁRAZOVÁ et al., 2008). For genotoxicity studies are plants highly responsible and 
sensitive. Their beneficial interest is that seeds and pollen grains can be easy storage 
away and offer cheap, relative easy and accurate toxicological assessment (KRISTEN, 
1997). By their ability to accumulate toxic substances, they indicate metal presence in 
the environment even in very low concentration (CHANDRA, 2004).  

Contamination of soil and water by Cr and Ni are of particular recent concern. The 
impact of Cr contamination on the physiology of plants depends on the metallic 
species responsible for its mobilization, uptake and toxicity in the plant system 
(BENNICELLI et al., 2004). While Cr is not considered an essential element for plant 
nutrition (SHARMA et al., 1995), Ni is classified an essential trace element (BROWN 
et al., 1987); and although it is found everywhere in the environment, it usually occurs 
only in trace amounts. 
 

2. Materials and methods 
 

Simultaneous phytotoxicity and mutagenicity assay was carried out on plant 
species Vicia sativa L. var. Klára according to MIADOKOVÁ et al. (2005). After 24 h 
of soaking at 25 °C in distilled water or solution with metal concentration equal to IC50 
value the seeds of V. sativa were allowed to germinate in Petri dishes (diameter = 18.5 



108                                                                                 Fargašová, A. and Lištiaková, J. 
 
cm) with filter paper soaked with the same concentration of tested metal as that used 
for soaking. Phytotoxicity was assayed after 72 h of the dark cultivation in the 
thermostat at 25 °C by the same way as described previously SVETKOVÁ and 
FARGAŠOVÁ (2007) for S. alba. The roots and shoots of V. sativa seedlings were 
measured and the growth inhibition percentage was assessed. The seedling roots used 
for chromosome and genome mutability evaluation were fixed and permanent slides 
were prepared by the Feulgen method. Chromosome aberrations were determined at 
least in 500-anatelophases. For statistic analysis the Student’s t-test was used.  

The procedures for maintaining the Tradescantia plants and for analyzing 
micronuclei frequency in the tetrads have been described by MIŠÍK et al. (2006; 
2007). Tradescancia paludosa clone 03 was standardly cultivated at the Department of 
Botany, Faculty of Natural Sciences, Comenius University in Bratislava. Inflorescence 
were harvested at the 8-10-buds stage and immersed into 500 ml of tested metal 
solutions (100 mg/l CrO3 and NiCl2, 1000 mg/l Cr(NO3)3) for 12 h. As control tap 
water was used. The 24 h reconvalescence, during which inflorescence peduncles were 
dipped in 500 ml of tap water, succeeded to 12 h exposure. Then the buds were fixed 
for 24 h in ethanol : acetic acid (3 : 1). The fixed material was stored in 70% ethanol. 
Slides were prepared from the fixed material using the aceto-carmine squash 
technique. Micronuclei were scored in the early tetrad stages of pollen mother cells. In 
the present study, 15 to 20 inflorescences comprised a sample. Three hundred tetrads 
were scored from each of five slides prepared from a treatment sample for a total of 
1,500 tetrads per plot. Data were recorded as the number of micronuclei (MCN) per 
100 tetrads. A change of frequency of MCN/100 tetrads was considered statistically 
significant (at P < 0.05) if the difference between the mean of the control population 
and the mean of the treated population was at least twice as large as the standard error 
of the difference between the two means (MA et al., 1994; MIŠÍK et al., 2007).  

For samples of tested metals NiCl2.6H2O, Cr(NO3)3.9H2O and CrCO3 of analytical 
grade p.a. were obtained from Lachema, Brno, Czech Republic. 

All experiments for growth inhibition were set up in a completely randomized 
design with three replicates. Chronic toxicity was assessed as inhibition of roots and 
shoots growth and results were evaluated by Gryck-Haustein method and IC25, IC50 
and IC75 concentrations were determined. The results were statistically evaluated by 
using Toxicity program.  
 

3. Results and discussion 
 

Toxic effects of heavy metals, mainly during chronic exposure, are not visible 
immediately hence ecotoxicological studies request also assessment of genotoxicity. 
Genotoxicological effect is developed then the concentration is low order than that for 
fytotoxicity effect (MIČIETA and MURÍN, 1998). For phytotoxicity and 
clastogenicity study V. sativa seedlings were used. Phytotoxicity was determined 
through IC25, IC50 and IC75 values and for roots and shoots the strongest inhibitory 
effect had Ni(II) (Fig. 1). No significant differences were confirmed between Cr(III) 
and Cr(VI) adverse effects on V. sativa shoot growth. On the basis of these values, and 
their statistical evaluation, metals can be arranged in the following rank orders of 
inhibition: for roots: Ni(II) > Cr(VI) > Cr(III); for shoots: Ni(II) > Cr(VI) ≥ Cr (III). 



Nova Biotechnologica 9-2 (2009)                                                                               109 
 

 
 

 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 

Fig. 1. IC25, IC50 and IC75 values and their 95% confidence intervals (CI) (ml/l) for Vicia sativa L. after 72 h 
application; the mean of three determinations with a standard deviation 6% or less 
 

For mutagenicity assay root tips of V. sativa were used and chromosome 
aberrations were determined at least in 500-anatelophases. All tested metals exerted in 
V. sativa a significant increase of chromosomal aberration rate in applied 
concentrations (Table 1). From all tested metals Cr(VI) invoked maximum of 
aberrations in anatelophase cells. The rank order of aberrations fall was: Cr(VI) > 
Ni(II) > Cr(III).  
 
Table 1. Potential clastogenicity evaluation of Cr and Ni in Vicia sativa L. (n = 500) 
 

Metal concentration (mg/l) 
Metal 

Ni Cr 
Number of 

aberrations ± SD 
Percentage of 

aberrations ± SD 
Control <0.07 <0.01 7 ± 0.69 2.33 ± 0.23 
Ni(II) 16.46 ± 2.16  10 ± 0.75 ** 3.33 ± 0.25 ** 
Cr(III  114.41 ± 13.36 8 ± 0.75 * 2.67 ± 0.25 * 
Cr(VI)  69.69 ± 8.66 13 ± 0.69 ** 4.33 ± 0.23 ** 

SD – standard deviation; ** significant differences in comparison with control at P <0.01; * significant 
differences in comparison with control at P <0.05; control – sterile distilled water  
  

Genetic variation in susceptibility to environmental agents and metals can be 
considered as differences in metabolism of these agents in various organisms 
(OMENN, 1991). In addition, DNA target size and DNA content are also important in 
determining genotoxic hazard of metals. As described KOVALCHUK et al. (1998) 
and CHAUHAN et al. (1998) genotoxicity can be obtained as a result of multipolar 
anaphase and c-mitose or damage of protein synthesis in the presence of DNA 



110                                                                                 Fargašová, A. and Lištiaková, J. 
 
toxicant. Simultaneous toxicity and clastogenity of wastes with Cr and Ni content was 
also confirmed for V. sativa  by MIADOKOVÁ et al. (1999) and for V. faba and 
Allium cepa by Chandra et al. (2004; 2005). Chromozomal fragments and bridges 
created in Cr(VI) presence indicated as introduced QUIAN (2004) that CrO3 affecting 
DNA structure and conformation. 
 
Table 2. Micronuclei frequency in the Tradescantia pollen tetrads after treatment with Cr and Ni solutions 
(n = 1,500) 
 

Metal concentration (mg/l) Metal 
Ni Cr 

Number of 
micronuclues ± SD 

Percentage of 
micronucleus ± SD 

Control <0.07 <0.01 43 ± 13.74 2.89 ± 0.92 
Ni(II) 24.71 ± 0.25  59 ± 17.48 3.93 ± 1.17 
Cr(III  130.00 ± 1.30 47 ± 16.61 3.13 ± 1.11 
Cr(VI)  52.00 ± 0.52 60 ± 15.98 4.00 ± 1.07 

SD – standard deviation 
 

For determination of Cr and Ni genotoxic effects was also used analysis of 
micronuclei frequency in the pollen  tetrads of Tradescantia plants. As it is evident 
from Table 2 none of tested metal significantly stimulated, in comparison with the 
control, micronuclei frequency and genotoxic effect was decreased in order: Cr(VI) ≥ 
Ni(II) > Cr(III). Tradescantia micronucleus test (Trad-MCN) belongs together with 
Allium cepa L. and Vicia faba L. tests with root tips to most frequently used 
genotoxicity tests on plants (MAJER et al., 2005) and it is very popular now for in situ 
biomonitoring of air pollution (MIŠÍK et al., 2006; 2007). Results obtained during our 
genotoxicity tests are in good agreement with those introduced by KNASMÜLLER et 
al. (1998) when CrO3, CrCl3 and NiCl2 up to concentration 10 mM did not evoke 
genotoxic effects. The same conclusion also introduced MAJER et al. (2005) for 
Cr(III).  Higher genotoxicity of Cr(VI) than Cr(III) determined during our experiments 
also described NĚMEČEK et al. (2002). ROSSMAN (1995) introduced that molecular 
mechanism of DNA damage by Cr(VI) involve induction of DNA-DNA and DNA-
protein cross-links and genotoxic effect can be also increased by reactive oxygen 
produced during intracellular reduction. For Ni(II) no genotoxic effects confirmed for 
bacteria ROSSMAN (1995) and PATIERNO and COSTA (1987) introduced that 
mutations after Ni applications are also the result of DNA damage and DNA-protein 
cross-links formation.   
 
Acknowledgements This study was supported by grants VEGA 1/4361/07, VEGA 
1/0238/08 and APVV – 0231-07. 
 

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