SKIN 
 

July 2022     Volume 6 Issue 4 
 

(c) 2022 THE AUTHORS. Published by the National Society for Cutaneous Medicine. 354 

SKIMages 
 

 

Hypopigmented Patches on the Trunk of a 25-year-old Hispanic 
Male 
 

McKenzie A. Dirr, BA, BS1, Nicholas Brownstone, MD2, Darrell Rigel, MD, MS3 

 
1 Medical University of South Carolina, Charleston, SC 
2 National Society for Cutaneous Medicine, New York, NY 
3 Department of Dermatology, Mount Sinai Icahn School of Medicine, New York, NY 
 

 
 

 
 

We present a case in which a 25-year-old 
Hispanic male presented to clinic with 
asymptomatic patches of hypopigmented 
skin on the arm and back. He first noticed the 
lesions two weeks prior, with no previous 
history of similar complaints. Upon clinical 
inspection, he was found to have Tinea 
Versicolor (TV) and treatment was promptly 
initiated. This demonstrates a case of 

hypopigmented TV in a patient with 
Fitzpatrick skin type IV, and illustrates an 
example of one of the many forms in which 
TV may present.  
 
Tinea Versicolor, or Pityriasis Versicolor, is a 
pathologic fungal colonization of the stratum 
corneum.1,2,3 Caused by Malassezia, a fungal 
genus that is normally found on the skin, TV 



SKIN 
 

July 2022     Volume 6 Issue 4 
 

(c) 2022 THE AUTHORS. Published by the National Society for Cutaneous Medicine. 355 

pathologically overgrows in warm, humid 
conditions. 1,2,3 TV is lipophilic and is 
commonly found in anatomic locations with 
increased density of sebaceous glands, such 
as the arms, trunk, neck, and face.1,2,3 While 
TV does not damage the underlying skin or 
cause long-term morbidity or mortality, it can 
present a frustrating cosmetic challenge for 
patients.1 

 
TV can have a varied presentation, including 
multicolored, well-demarcated lesions of 
hypopigmentation, hyperpigmentation, and 
erythematous macules that may scale, itch, 
or present asymptomatically. 1,2 The etiology 
of hypopigmentation is multifactorial, 
including Malassezia-induced reduction of 
melanosome size, invasion of keratinocytes 
which ultimately damages melanocytes and 
blocks formation of melanin, and blockade of 
UV light, causing unevenness in 
pigmentation.1,3 Inflammatory reactions and 
enlargement of melanosomes can lead to 
hyperpigmentation.1,2,3  

 
The varied presentation can lead to a delay 
in diagnosis, and as such, it is import to 
differentiate how TV may present in patients 
of different Fitzpatrick skin types. It had 
previously been proposed that in patients 
with skin of color, hypopigmentation is more 
common, with the potential to combine into a 
larger, amalgamated area of discoloration, 
while patients with lighter skin tones are more 
likely to present with hyperpigmentation. 1,4 
However, in a study done by Aljabre et. al, 
this claim was not supported, with the authors 
finding that patients with skin of color are 
likely to present with any TV variation, 
including macules of dark brown or gray-
black hyperpigmentation.1,2,4 Further 
publications have also supported the finding 
that patients with skin of color are likely to 
develop any TV variation, and thus it is 
important for physicians to have an increased 

understanding of all of the possible TV 
presentations.2  
 
The diagnosis of TV can be made clinically, 
with biopsy, using Wood’s light, or via KOH 
prep and direct visualization under light 
microscopy, where it is visualized as non-
branching pseudohyphae with circular 
spores. 1,2,3 Management of TV in patients 
should take into account patient preference 
and disease course, as a topical antifungal is 
sufficient in most cases. 1,2 However, in 
severe or persistent infections, oral 
antifungals may be the more effective 
solution.1,2 It is important to note the 
increased risk of post-inflammatory 
hyper/hypopigmentation seen in patients with 
skin of color, which may warrant an 
aggressive treatment approach, while 
simultaneously weighing the potential side 
effects of oral medication.1,2   
 
Conflict of Interest Disclosures: None 
 
Funding: None 
 
Corresponding Author: 
McKenzie A. Dirr, BA, BS 
Medical University of South Carolina 
96 Jonathan Lucas Street 
Suite 601, MSC 617 
Charleston, SC 29425  
Phone: 843-792-2081 
Email: dirr@musc.edu 

 
 
References: 
1. Vashi NA, Maibach HI. Dermatoanthropology of 

Ethnic Skin and Hair. 1st ed. 2017. (Vashi NA, 
Maibach HI, eds.). Springer International 
Publishing; 2017. doi:10.1007/978-3-319-53961-
4 

2. Kallini JR, Riaz F, Khachemoune A. Tinea 
versicolor in dark-skinned 
individuals. International journal of dermatology. 
2014;53(2):137-141. doi:10.1111/ijd.12345 

3. Schwartz RA. Superficial fungal infections. 
Lancet. 2004;364(9440):1173–82. 

4. Aljabre SH, et al. Pigmentary changes of tinea 
versicolor in dark-skinned patients. Int J 
Dermatol. 2001;40(4):273–5. 

mailto:dirr@musc.edu


SKIN 
 

July 2022     Volume 6 Issue 4 
 

(c) 2022 THE AUTHORS. Published by the National Society for Cutaneous Medicine. 356