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Introduction

“Swimmer’s ear” or acute otitis externa is a term that is used 

commonly by medical practitioners and patients. It refers to 

an inflammatory condition of the external auditory canal, with 

anatomical regions which may stretch distally to the pinna and 

proximally to the tympanic membrane of the ear.1 The prevalence 

in swimmers is approximately five times more than that in non-

swimmers, with an annual incidence of approximately 1% and 

an overall lifetime incidence of 10%.2 It occurs mostly during 

spring and summer, with age peaks at 7–12 years, and decreases 

after the age of 50 years. Continuous or prolonged exposure to 

water dissolves oil and wax in the ear canal, which results in dry, 

fissuring skin, thereby effectively obliterating the ear’s natural 

defense mechanism against microorganisms. Minor trauma 

from vigorous or inappropriate ear cleaning could also be an 

associated cause.

Swimmer’s ear presents with a sudden onset of otalgia, pruritis, 

canal oedema and erythema, and is often accompanied 

by otorrhea and hearing loss. A classical finding of severe 

tenderness with movement of the pinna is characteristic of this 

condition. Furthermore, it can be classified as mild (minimal 

canal oedema with minor discomfort and pruritis); moderate 

(partly occluded canal with intermediate pain and pruritis); and 

severe (completely occluded ear canal with intense pain, fever, 

erythema and lymphadenopathy).3  

Bacterial causes are responsible for the majority of acute cases.  

A recent study conducted in South Africa showed that 

Pseudomonas aeruginosa (33%) and Staphylococcus aureus 
(24%) were the main organisms responsible for otitis externa.4 

Proteus mirabilis (5%) and fungal elements (5%) were identified 
as additional culprits. This correlates well with the findings 

in the literature from international studies of a prevalence of 
P. aeruginosa of 38%, and S. aureus of 17%.5 However, a third 

of all cases are attributed to polymicrobial Gram-positive 

and Gram- negative colonisation, which includes Escherichia 
coli, Enterobacter cloacae, Haemophilus influenzae, Klebsiella 
pneumoniae and Alcaligenes faecalis. Viral infections (herpes 
simplex and herpes zoster) account for a much smaller 

percentage.6 

Non-infective causes are less frequent and present as chronic 

otitis externa. These include allergic contact dermatitis (e.g. 

secondary to otic agents and soap), eczematous dermatitis (atopic 

dermatitis), irritant contact dermatitis, psoriasis, seborrheic 

dermatitis, acne vulgaris and systemic lupus erythematosus.

Clinical pharmacology and practice guidelines 

Various guidelines for the management of acute otitis externa 

are published and updated on a regular basis by different 

otolaryngology authorities worldwide.1 A shared commonality 

in these guidelines includes the appropriate diagnosis of 

acute otitis externa, the provision of adequate pain relief, and 

the rational prescribing of topical or systemic antibiotics. In 

addition, the treatment should include cleaning the ear canal, 

avoiding aggravating factors (protecting the ear from additional 

moisture or further mechanical injury), and considering alternate 

diagnoses in unresponsive cases.7  

Topical anti-infective agents

First-line therapy with antiseptics is still preferred to antibiotics 

in uncomplicated and mild cases of local external ear infections.8 

These agents possess bacteriostatic and fungistatic properties, 

and render the ear canal less habitable to microorganisms. In 

addition, they may also loosen impacted debris, and reduce 

swelling and inflammation (when present) in mild cases of 

swimmer’s ear. Various acidifying ear drop formulations can 

be used, which are normally freshly prepared by a pharmacist. 

These formulations include 1% acetic acid in distilled water, 1% 

“Swimmer’s ear” or acute otitis externa is a common condition involving the exterior part of the ear, including the ear canal and the 

pinna. Inflammation and pain are the main features, with bacterial infection mainly due to Staphylococcus aureus and Pseudomonas 
aeruginosa. It can easily be treated in its uncomplicated stage, and the symptoms usually do not last for more than 72 hours. 
Management ranges from primary care to a specialist otolaringological intervention. Several nonpharmacological treatments, in 

addition to various pharmacological options, are employed in the treatment and prevention of swimmer’s ear. These treatments 

range from simple, adequate and appropriate ear cleaning, to topical antibiotics, corticosteroids and analgesics. Severe cases may 

require systemic antibiotics, oral anti-inflammatory drugs and even opioid analgesics.

Keywords:  acidifying antiseptics, antibiotics, corticosteroids, otitis externa, swimmer’s ear

S Afr Fam Pract
ISSN 2078-6190   EISSN 2078-6204 

© 2015 The Author(s)

REVIEW

South African Family Practice 2015; 57(5):4-8
 
Open Access article distributed under the terms of the 
Creative Commons License [CC BY-NC-ND 4.0] 
http://creativecommons.org/licenses/by-nc-nd/4.0

Clinical evidence in the management of swimmer’s ear
Andre Marais*

Department of Pharmacology, School of Medicine, Faculty of Health Sciences, University of Pretoria, South Africa
* Corresponding author, email: dramarais@gmail.com, andre.marais@up.ac.za



S Afr Fam Pract 2015;57(5):4-86

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acetic acid in 90% alcohol and distilled water, and 2% boric acid 

in 90% alcohol and distilled water. Clioquinol (3%) in cream or an 

ointment base can be used as an alternative to the droplet form. 

The use of acidifying antiseptic agents should be avoided if the 

tympanic membrane is known or suspected to be non-intact. 

Skin irritation, hypersensitivity reactions and contact dermatitis 

are the most common side-effects of locally applied antiseptic 

agents. Therapy should be continued for a week, after which 

those patients whose symptoms have not resolved should be 

changed to a combination antibiotic-corticosteroid regimen. The 

preventative use of topical acidifying agents or 70% alcohol can 

also be considered in children who are prone to swimmer’s ear.9 

Topical antibiotics

Although topical antibiotics have a very high efficacy of 

between 29% and 63% in the treatment of otitis externa, their 

routine and indiscriminant use should be avoided.10  There is 

increasing concern about resistance and potential treatment 

failure, especially when these agents are used chronically.11 

Topical antibiotics providing coverage against P. aeruginosa 
and S. aureus should be considered in cases when there is 
only mild inflammation with obvious infection. Quinolones 

(ofloxacin and ciprofloxacin) are equally effective compared to 

the aminoglycosides (tobramycin and gentamycin) in treating 

P. aeruginosa and S. aureus, whereas neomycin only exhibits 
activity against S. aureus, and polymixin B against P. aeruginosa. 
Neomycin is commonly combined with polymixin B in otological 

preparations for this reason.

It is advisable to avoid the use of aminoglycosides in cases where 

a non-intact tympanic membrane is present as these agents are 

known to be a potential cause of iatrogenic hearing loss and 

balance dysfunction. It was shown in a recent retrospective 

study in the USA that aminoglycosides were responsible for 

47% of civil litigation cases, where drugs were implicated as the 

causative factor.12 The fluoroquinolones are more suitable when 

tympanic membrane perforation is suspected, but may cause 

local irritation and allergic dermatitis.13

Systemic antibiotics

Systemic antibiotics are only indicated in severe cases of otitis 

externa, when the infection has spread beyond the external ear 

canal, or signs of deep tissue infection (cellulitis) are present. 

Oral therapy, with ciprofloxacin and/or ceftazidime, for at least 

10 days, is recommended. Systemic therapy should be given for 

3–6 weeks in cases of necrotising external otitis.14 

Topical corticosteroids

Topical glucocorticoids decrease pain and inflammation. These 

agents are useful in reducing pruritis and swelling in the absence 

of a suspected infection. Therefore, they are used to control 

eczematous conditions of the external ear, including otitis 

externa. Various corticosteroids are available as either topical 

drops or in cream and ointment form. The most commonly 

used otological topical corticosteroids include hydrocortisone, 

prednisolone, betamethasone, dexamethasone and flumethas-

one (in order of increasing potency).8 These topical agents are 

generally well tolerated, but should not be used in the presence 

of viral of fungal infections, especially in children, as the risk of 

exacerbating the condition is increased.9

Antibiotic-corticosteroid combinations

Several combination antibiotic-corticosteroid preparations are 

available. However, it was indicated in a recent meta-analysis 

that none of the specific combination therapies was superior to 

the rest.8 The presence of severe inflammation with infection and 

dermatitis necessitates the use of these agents. Their use should 

be limited and discouraged in the absence of a proper diagnosis. 

Analgesics

Topical analgesics and local anaesthetics are of limited use, and 

might only demonstrate some benefit in mild cases of the disease. 

Oral nonsteroidal anti-inflammatory drugs (NSAIDs), such as 

ibuprofen, still remain the most effective way of managing pain 

associated with otitis externa. Opioid analgesics are reserved for 

patients with severe pain which has not responded to topical 

treatment or systemic NSAIDS.

Currently available topical preparations in South Africa for the 

treatment of otitis externa are detailed in Table I. 

Conclusion and recommendations

“Swimmer’s ear” or acute otitis externa is a common condition that 

affects many people annually. The treatment of uncomplicated 

Table I: Currently available topical preparations in South Africa for the treatment of otitis externa

Composition Dosage Pack size SEP

Topical anti-infective agents

Compounded mixtures Acetic acid 1% 4-6 drops, 2 times daily 100 ml N/A

Acetic acid 1% in 90% alcohol 100 ml

Boric acid 2% in 90% alcohol 100 ml

Clioquinol 3% 100 g

Topical antibiotics

Ciloxan® (Alcon Laboratories) Ciprofloxacin 3 mg/ml 2 drops, 4 hourly 5 ml R147.76

Exocin® (Allergan) Ofloxacin 3 mg/ml 1 drop, 4 times daily 5 ml R68.35

Octin® (Cipla Medpro) 5 ml R53.18

Tobrex® (Alcon Laboratories) Tobramycin 3 mg/ml 2 drops, 4 hourly 5 ml R98.66



Clinical evidence in the management of swimmer’s ear 7

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conditions can be managed at primary care level. Cleaning of the 

ear canal, including the removal of impacted cerumen, is the first 

step in its management. This facilitates healing, and improves 

the penetration of eardrops into the area of inflammation.15 

Topical therapy with non-antibiotic preparations containing 

acidifying agents, with or without corticosteroids, is indicated for 

mild cases. Moderate and severe disease warrants the additional 

inclusion of a topical antibiotic, while the placement of a wick 

Topical corticosteroids

Maxidex® (Alcon Laboratories) Dexamethasone 1 mg/ml 3-4 drops, 2-4 times 
daily

5 ml R133.19

Betnesol® (Aspen Pharmacare) Betamethasone sodium phosphate 
0.1%

2-3 drops, 2-3 hourly 5 ml R148.71

Dilucort® (Aspen Pharmacare) Hydrocortisone 0.5% Apply twice daily 25 g cream R30.16

Bilocort® (Akacia) Hydrocortisone 1% 25 g cream R9.91

Procutan® (MSD) 20 g cream R203.70

Stopitch® (Al Pharm) 20 g cream R24.41

Adco-Betamethasone® (Al Pharm) Betamethasone valerate 0.1% Apply 1-3 times daily 15 g cream R15.42

Betnovate® (Sekpharma) 15 g cream
30 g cream

R27.24
R28.48

Lenovate® (Aspen Pharmacare) 15 g cream
500 g cream

R18.81
R627.34

Persivate® (Aspen Pharmacare) 15 g cream
500 g cream

R15.39
R513.09

Advantan® (Bayer) Methylprednisolone 0.1% Apply 1-3 times daily 20 g cream and ung
50 g cream and ung

R146.35
R365.87

Antibiotic-corticosteroid combinations

Sofradex® (Sanofi-Aventis) Framycetin sulphate 5 mg/ml 2-3 drops, 3-4 times 
daily 

8 ml R120.09

Gramicidin 0.05 mg/ml

Dexamethasone 0.5 mg/ml

Phenyletanol 0.5%/ml

Otosporin® (Aspen Pharmacare) Polymixin B sulphate 10 000 u/ml 3-4 drops, 3-4 times 
daily

10 ml R93.18

Neomycin sulphate 
3 400 u/ml

Hydrocortisone 10 mg/ml

Cilodex® (Alcon Laboratories) Ciprofloxacin 3 mg/ml 4 drops, 2 times daily 5 ml R204.10

Dexamethasone 1 mg/ml

Betnesol-N® (Aspen Pharmacare) Betamethasone sodium phosphate 
0.1%

2-3 drops, 2-3 hourly 5 ml R118.21

Neomycin sulphate 0.35% 10 ml R236.40

Locacorten Vioform Eardrops® (Pharmaco) Flumethasone pivalate 0.2 mg/ml 2-3 drops, 2 times daily 7.5 ml R131.00

Clioquinol 10 mg/ml

Maxitrol® (Alcon Laboratories) Polymyxin B 6 000 u/ml
Dexamethasone 1 mg/ml
Neomycin 3500 u/ml

1-2 drops, 6 times daily 5 ml R137.14

Apply 3-4 times daily 3.5 g ung R140.67

Tobradex® (Alcon Laboratories) Dexamethasone 1 mg/ml 1 drop, 4 hourly 5 ml R153.50

Tobramycin 3 mg/ml 3.5 g ung

Topical analgesics

Aurone® (Aspen Pharmacare) Phenazone 50 mg/ml 5-10 drops, 2 hourly 15 ml R46.57

Covancaine® (Al Pharm) Sulphacetamide sodium 100 mg/ml 5 drops, 1-4 hourly 20 ml R41.17

Phenazone 50 mg/ml

Benzocaine 10 mg/ml

Urea 120 mg/ml

Adco-Otised® (Al Pharm) Phenazone 50 mg/ml 2-3 drops, 4 hourly 15 ml R8.41

Benzocaine 10 mg/ml

Glycerine 1.185 g/ml

N/A: not applicable, SEP: single exit price [as listed in the Monthly Index of Medical Specialties. 2015;55(4)], ung: ointment



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should be considered in severe cases only. This will allow 

medication to reach the medial aspect of the ear canal. Systemic 

antibiotics should only be prescribed in immunosuppressed 

patients, or when a deeper infection spreading beyond the 

ear canal is present. The use of systemic NSAIDs is preferred to 

topical analgesics and local anaesthetics owing to their limited 

efficacy. All cases of malignant or necrotising otitis externa 

should be referred to an otolaryngologist.16

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