Introduction

Local cryotherapy is commonly used in the management of  
acute muscle injuries.

14,20,24,25
 Despite this, the clinical ben-

efits of  cryotherapy for acute muscle injuries are still ques-
tioned, because of  the lack of  suitable randomised controlled 
studies.

3,12

Cooling can be achieved using different modalities such 
as wet ice, crushed-ice packs, dry ice, frozen gel packs, 
endothermic chemical reaction packs, refrigerant cooling 
devices, coolant sprays, cold water baths, cooling blankets, 
cold air and electrical cooling devices.

2,5,22,24,33
 Many of  

the physiological effects of  local cooling have been well 
described. Cooling reduces metabolic rate,

10,26,31
 local  

adenosine triphosphate utilisation,
34 

bloodflow,
4,10,23

 the 
inflammatory response

23
 and oedema formation.

6,11
 Cooling 

also has an analgesic effect, slows nerve conduction velocity, 
and decreases muscle spasm and spasticity.

1,15,18,19,28

There is still controversy over what constitutes the optimal 
modality, duration and frequency of  cryotherapy for muscle 
injury.

3
 The temperature of  skin, subcutaneous tissue and 

muscle has been measured at various depths below the 
skin before and after different forms of  cryotherapy,

2,5,7,14 

and adipose tissue has been shown to have an insulating 
effect, impairing cooling of  the underlying muscle.

27,29
 In 

resting muscle, there is normally a temperature gradient, 
with temperature increasing with depth.

7,16,32
 This gradient is 

altered by cryotherapy.

To date, studies on the use of  cryotherapy have only 

orIgInal research arTIcle

The effect of icepack cooling on skin and muscle tempera-
ture at rest and after exercise 

Maurice Mars1 (MB chB, MD) 

Brian hadebe2 (MBchB) 

Mark Tufts3 (Msc) 
1
Department of  TeleHealth, Nelson R Mandela School of  Medicine, University of  KwaZulu-Natal, Durban

2
Postgraduate student, private practitioner

3
Department of  Physiology, Faculty of  Health Science, University of  KwaZulu-Natal, Durban

abstract

objective. To compare cooling of  skin, subcutaneous fat 
and muscle, produced by an icepack, at rest and after 
short-duration exhaustive exercise.

Methods. Eight male subjects were studied. With the 
subject supine, hypodermic needle-tip thermistors were 
inserted into the subcutaneous fat and the mid-portion of  
the left rectus femoris, to a depth of  1 cm plus the adipose 
thickness at the site, and a temperature probe was placed 
on the skin overlying the needle tips. A pack of  crushed 
ice was applied for 15 minutes and temperatures were re-
corded before, during, and for 45 minutes after icepack 
application. Thereafter, subjects underwent a ramped, 
treadmill, VO2max test, an icepack was applied after tem-
perature probes were inserted into the right leg and mea-
surements were made as before. 

results. After the treadmill run, skin (Sk), subcutaneous 
(SC) and muscle (Ms) temperatures (mean ± standard de-
viation (SD)) were 0.9 ± 1.3, 1.0 ± 0.7 and 1.3 ± 0.8°C 
higher than at rest. After 15 minutes of  icepack cooling, 
temperatures fell in the exercised limb by 22.7 ± 1.5°C 
(Sk), 13.5 ± 4.2°C (SC) and 9.3 ±  5.5°C (Ms) and in the 
control limb by 20.7 ± 2.9°C (Sk), 11.4 ± 2.0°C (SC) and 
8.7 ±  2.6°C (Ms). The reductions in temperature were sig-
nificant in both the control and exercised limbs. Forty-five 

corresPonDence:

Maurice Mars
Dept of  Telehealth
Nelson R Mandela School of  Medicine
University of  KwaZulu-Natal
Pvt Bag 7
Congella 
4013
Tel: 031-260-4543
Fax: 031-260-4737
E-mail mars@ukzn.ac.za

minutes after icepack cooling, muscle temperature was 
still approximately 5°C lower in both the rested and exer-
cised muscle (p < 0.001). Individual variations in response 
to cooling were noted. 

conclusions. Cooling of  superficial muscle occurs after 
high-intensity exercise. The degree of  cooling is not uni-
form. This may be due to differences in the sympathetic 
response to cooling, influencing haemodynamic and ther-
moregulatory changes after exercise. This needs further 
investigation.

60                                 saJsM  vol 18  no. 3  2006

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62               saJsM  vol 18  no. 3  2006

investigated subjects at rest. In the athletic setting, it is 
common to apply an icepack to an injury as soon as the athlete 
stops competing or training. Depending on the intensity and 
duration of  exercise, the athlete’s muscle temperature, core 
temperature and cardiac output, will have increased.

8,16 

These haemodynamic and temperature changes may affect 
temperature flux under a cooling pack. 

The aim of  this study was to investigate the effect of  
an icepack on muscle cooling following acute exercise and 
compare it with muscle cooling in the rested state.

Methods

The study was undertaken with the approval of  the Biomedi-
cal Ethics Committee of  the University of  KwaZulu-Natal. 
Eight adult male volunteers who all exercise on a regular 
basis, participating in at least four soccer practices and/or 
matches a week, were studied and all signed informed con-
sent. At a preliminary visit they underwent medical screen-
ing and were familiarised with the apparatus and the testing 
procedures.  

On the day of  testing, subjects lay supine for 15 minutes 
to acclimatise to the ambient temperature of  the laboratory. 
They were dressed in running shorts and an athletic vest. The 
site for measurement of  temperatures was determined as the 
point halfway between the anterior superior iliac spine and 
the superior pole of  the patella. The skinfold thickness was 
measured at this point using skin callipers (John Bull skinfold 
callipers, British Indicators, England) and the thickness of  
adipose tissue was taken as half  the skinfold thickness. 

A cutaneous temperature probe (YSI 4494, Yellow Springs 
Instrument Co) was attached to the skin over the middle of  
the left thigh in the anterior midline. After cleaning the skin 
with alcohol, and using aseptic techniques, appropriately 
sterilised 22G hypodermic, needle-tip thermistors, (YSI Inc 
Precision 5510 series) were inserted into the subcutaneous 
fat and to a depth of  adipose thickness plus 1 cm into the 
quadriceps muscle, so as to lie beneath the skin temperature 
probe. The needles were inserted from the lateral aspect 
of  the thigh such that the barrel of  the hypodermic needle 
was not under the icepack and the needles were taped to 
the skin to prevent movement. A needle guide was used to 
assist needle placement at the correct depth in the muscle.  
The temperature probes and hypodermic thermistors were 
attached to a YSI 4000A thermometer and each channel 
was calibrated using the unit’s selfcalibration. The needles 
were cleaned and sterilised according to the manufacturer’s 
instruction after each use.

 A 20 cm x 10 cm x 5 cm pack of  crushed ice in a wet towel 
was then placed longitudinally over the temperature probe 
and left in situ for 15 minutes. The icepack was not strapped 
to the subject and no compression other than the weight of  
the icepack was applied. Temperature measurements were 
recorded every minute, for 5 minutes before application of  
the icepack, during the ice application and for 45 minutes 
after removal of  the icepack. Thereafter the hypodermic 

needle probes and skin temperature probe were removed. 

An hour later subjects underwent a ramped VO2max test on 
a treadmill using a previously described method.

21
 Oxygen 

consumption and carbon dioxide production was measured 
using open circuit spirometry (Oxycon Champion, version 
4.3 – CE 0434, Jaeger). The spirometer was calibrated daily. 
On completion of  the treadmill run, subjects immediately lay 
supine on a plinth and the temperature probes were attached 
and inserted into the right leg as previously described. 
After 5 minutes, an icepack was applied for 15 minutes and 
recordings were made as before. 

Data are expressed as the mean and one standard 
deviation and the 95% confidence interval is given where 
appropriate. The rate of  change of  temperature per minute 
was determined by calculating the difference between 
successive measurements, at 5 minute intervals (δy), and 
dividing this by 5 (δx).

Statistical analysis of  the differences of  means was by 
paired t-test and the differences of  means within and between 
groups by two-way repeated-measures ANOVA with post hoc 
testing using the Bonferroni test. Alpha was set at 5%.

results

The subjects’ mean ages, heights and weights were 21.3 ± 
3.0 years, 172.5 ± 6.6 cm and 61.8 ± 10.2 kg. The adipose 
thickness of  the right thigh was 0.43 ± 0.15 cm and the left 
0.43 ± 0.17 cm. The ambient temperature during the study 
was 21.3 ± 1.7 °C. Maximum oxygen consumption was 49.1 
± 4.2 ml.kg

-1
.min

-1
 and this was achieved at a respiratory ex-

change ratio of  1.15 ± 0.1. Treadmill running time was 12.2 
± 1.1 min.

The changes in skin, subcutaneous tissue and muscle 
temperatures are shown in Table I and Fig. 1. Exercise 
elevated skin and subcutaneous muscle temperatures. The 

TaBle I. skin, subcutaneous tissue and muscle tem-
peratures in rested and exercised limbs, recorded 5 
minutes after starting measurement (the time point 
immediately prior to the application of the icepack), 
at the end of cooling (20 min) and after 45 minutes of 
recovery (65 min), expressed as the mean ± standard 
deviation and the 95% confidence interval 

Time control °c 95% cI exercise °c 95% cI

Skin 5 min 28.8 ± 1.4 27.6 – 30.0  29.7 ± 1.3 28.6 – 30.8

Skin 20 min 8.1 ± 2.7 5.8 – 10.4 7.0 ± 1.2 6.0 – 8.0

Skin 65 min 25.6 ± 1.3 24.5 – 26.6 26.4 ± 1.5 25.1 – 27.6

SC 5 min 33.1 ± 1.1 32.2 – 34.0 34.2 ± 1.1 33.3 – 35.0

SC 20 min 21.7 ± 2.2 19.9 – 23.5 20.7 ± 5.0 16.5 – 24.9

SC 65 min 28.1 ± 1.0 27.3 – 29.0 29.5 ± 2.2 27.7 – 31.4

Ms 5 min 34.9 ± 1.2 33.9 – 35.9 36.3 ± 1.1 35.3 – 37.2

Ms 20 min 26.2 ± 3.5 23.3 – 29.1 27.0 ± 7.4 21.6 – 32.3

Ms 65 min 29.4 ± 1.1 28.4 – 31.4 31.0 ± 3.6 28.8 – 33.1

SC = subcutaneous tissue; Ms = muscle.

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64               saJsM  vol 18  no. 3  2006

application of  the icepack after exercise resulted, on average, 
in greater cooling of  skin, subcutaneous tissue and muscle, 
with the skin and subcutaneous tissue temperatures falling 

to below that of  the rested limb and then rewarming slightly 
faster, so as to be higher than the temperatures of  the rested 
limb 45 minutes after removal of  the icepack. The average 
temperature of  muscle after exercise did not fall below that 
achieved in the rested limb during cooling.

This response was not constant and there were individual 
variations, with some subjects showing a marked reduction in 
muscle temperature after cooling (Fig. 2) and others showing 
only minimal cooling of  muscle after exercise (Fig. 3).

The time to maximum cooling varied between subjects but 
was similar for each subject. The average time to maximum 
cooling of  subcutaneous tissue after application of  the 
icepack was 15.1 ± 0.8 min, (95% CI: 15.5 - 16.7) at rest 
and 16.6 ± 0.7 min (95% CI: 16.1 - 17.1) after exercise and 
in muscle 20.1 ± 4.5 min (95% CI: 17.0 - 23.2), range 15 - 29 
min at rest and 20.4 ± 3.8 min (95% CI: 17.7 - 23.0), range 
16 - 28 min, after exercise (Fig. 1). The differences were not 
statistically significant.

Skin, subcutaneous and muscle temperatures were 
compared at three fixed time points, 5 minutes after 

Fig. 1. Mean skin, subcutaneous (SC) and muscle (Ms) 
temperatures (°C) measured every minute, before, during 
and after application of  an icepack in the rested (c) and 
exercised (e) limb. The arrows represent the period that 
the icepack was applied. 

Fig. 3. Muscle temperature recorded every minute be-
fore, during and after application of  an icepack in rested 
(Ms(c)) and exercised muscle (Ms(e)) in subject 4. The ar-
rows represent the period that the icepack was applied.

Fig. 2. Muscle temperature recorded every minute, be-
fore, during and after application of  an icepack in rested 
(Ms(c)) and exercised muscle (Ms(e)) in subject 1. The ar-
rows represent the period that the icepack was applied. 

Fig. 5. The average rate of  change of  temperature per 
minute of  skin, subcutaneous tissue (SC) and muscle 
(Ms) at rest (c) and after exercise (e). 

Fig. 4. The absolute change in temperature (°C) from the 
start of  cooling at 5 min and subsequent measurements, 
of  skin (Sk) subcutaneous fat (SC) and muscle (Ms) at 
rest (c) and after exercise (e). Cooling occurred between 
5 min and 20 min.

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saJsM  vol 18  no. 3  2006                                                                                                                      65

commencing measurement (that is, just prior to the application 
of  the icepack), at the end of  icepack application and after 45 
minutes of  recovery (Table I). 

Five minutes after the treadmill run, skin, subcutaneous 
and muscle temperatures were 0.9 + 1.3°C, 1.1 + 0.8°C and 
1.3 + 0.8°C higher than at rest. After 15 minutes of  icepack 
cooling, temperatures fell in the exercised limb by 22.7 + 
1.5 °C (skin), 13.5 + 4.2°C (subcutaneous) and 9.3 + 5.5°C 
(muscle) and in the rested limb by 20.7 + 2.9°C, (skin), 11.4 
+ 2.0°C (subcutaneous) and 8.7 + 2.6°C (muscle). Forty-
five minutes after removing the icepack the reduction in 
temperature in the exercised leg was 3.3 + 1.3°C (skin), 4.6 
+ 1.4°C (subcutaneous) and 5.3 + 1.6 °C (muscle) and in the 
control limb, 3.2 + 0.8°C (skin), 5.0 + 0.7°C (subcutaneous) 
and 5.5 + 0.9°C (muscle). The fall in temperature after 15 
minutes of  icepack cooling and after 45 minutes of  recovery 
was significant, p < 0.001 for both the rested and exercised 
limb, as was the residual reduction in temperature after 45 
minutes of  recovery, p < 0.001. No differences in cooling 
were noted between the rested and exercised limbs.

As exercise raised skin, subcutaneous and muscle 
temperature, the absolute changes in temperature produced 
by cooling and rewarming were referenced to the temperatures 
at the commencement of  the icepack treatment (5 min) (Fig. 
4). The fall in temperature from before application of  the 
icepack to the end of  cooling (20 min) was significant for 
both rested and exercised limbs, p < 0.001, as was the fall in 
temperature between the commencement of  icepack cooling 
and the end of  the recovery period (65 min) p < 0.001. No 
differences in cooling were noted between the rested and 
exercised limbs. 

The rate of  change of  temperature per minute is shown 
in Fig. 5. No difference in the rate of  change of  temperature 
was noted between cooling and rewarming at rest or after 
exercise.

Discussion

The main finding of  this study is that cooling of  superficial 
muscle in response to an icepack is, on average, similar at 
rest and after exercise but that there are individual variations. 
This variability suggests that the haemodynamic and thermal 
alterations associated with intensive exercise may influence 
thermal flux at the depths measured, in some individuals.

During exercise, muscle temperature rises in response to 
increased metabolic activity, with a resultant increase in core 
temperature and arterial and venous blood temperature.

9,16
 

Associated with this is an increase in cardiac output, circulating 
the warmed blood more rapidly around the body. Cooling 
is based on the second law of  thermodynamics, with heat 
being transferred from a warmer body to a cooler body. For 
a muscle to cool, heat must be lost from muscle to adjacent 
cooler muscle tissue or subcutaneous fat. Subcutaneous 
fat will in turn lose heat to skin and finally heat will be 
dissipated by conduction to the icepack, and by radiation and 
evaporation to the atmosphere. The efficacy of  conduction to 

the coolant is dependent on the surface area being cooled 
and the physical properties of  the coolant. If  arterial blood is 
warmer than cooling muscle, then the muscle will also gain 
heat from the arterial blood, thereby assisting in lowering core 
temperature.  At rest there is a temperature gradient in large 
skeletal muscles, with the muscle tissue nearest the main 
feeder artery being the warmest and the most superficial 
muscle tissue, the coolest.

16,30
 During exercise this gradient 

is reduced, and after exercise it would be expected that as 
muscle cooling takes place, the gradient is re-established. 

While the rate of  cooling was similar before and after 
exercise, there was a trend for absolute cooling of  skin, 
subcutaneous tissue and muscle to be greater after exercise 
than at rest, with skin and subcutaneous tissue, which was 
warmer after exercise, being cooler than resting skin and 
subcutaneous tissue at the end of  the cooling period. Similarly 
the fall in muscle temperature was greater after exercise, but 
did not fall below that of  resting muscle. 

While the average data show a very similar response to 
cooling at rest and after exercise, the individual responses 
were not all the same. Similar variability has been reported in 
previous studies on rested muscle.

5,13,32,33
 The variability has 

been attributed to the insulating effect of  different thicknesses 
of  adipose tissue, and individual differences in sympathetic 
response to local cooling. In this study another possibility is 
that the needles were not all placed at the correct depths, 
although every precaution was taken to ensure that they were 
placed correctly.

The sympatho-adrenal system is activated by exposure 
to an external cold stimulus, and skin cooling in rats has 
been shown to elicit different sympathetic responses, 
dependent on the rate of  cooling.

17
 On the one hand, 

rapid skin cooling evokes a significant increase in plasma 
catecholamines with a reduction in skin catecholamines. The 
fall in skin catecholamines is attributed to their local release 
to cause vasoconstriction. Dermal vasoconstriction with an 
associated increase in plasma catecholamines may result in 
muscle arteriolar vasoconstriction with a more rapid fall in 
muscle temperature as seen in Fig. 2. On the other hand, 
slower cooling is not associated with an increase in plasma 
catecholamine concentration or a fall in skin concentration. 
With a reduction in local vasoconstriction, skin and muscle 
cooling would be slower as in Fig. 3. The catecholamine 
response to intense exercise may also play a role and this 
needs further investigation. It does not however appear to 
influence the timing of  the local cooling response as the time 
to maximum cooling in rested and exercised muscle, while 
varying between subjects, was relatively constant for each 
subject. Differences in exercise-induced catecholamine 
response may however account for the inter-subject variability 
in muscle cooling. Cooling of  skin causes a variable response 
in deeper tissues.  The implications of  this are that those 
who have very rapid skin cooling, while benefiting from the 
concomitant cooling of  muscle, are potentially at risk of  
developing ice burns and those with a muted response are 
unlikely to derive the benefits expected from muscle cooling. 

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66               saJsM  vol 18  no. 3  2006

What is required is a simple test that will elucidate who will 
have a rapid cooling response and who will have a slower and 
less effective response.

This study shows that cooling of  superficial muscle occurs 
after high-intensity exercise to exhaustion. The degree 
of  cooling is not uniform. This may be due to individual 
differences in the sympathetic response to cooling, which 
influence haemodynamic and thermoregulatory changes 
after exercise, although this was not measured in this study. 
Further studies are required to evaluate the sympathetic 
response to cooling after exercise and the temperature 
changes in deeper muscle tissue after exercise of  different 
intensity and duration. 

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