SAJSM 603 (CASE REPORT).indd


CASE REPORT

SAJSM  VOL. 27  NO. 1  2015   23

Primary spontaneous pneumothorax (PSP) is relatively uncommon in the athletic population. Because of the subtle nature of the 
symp toms, the diagnosis is easily missed, which can lead to unnecessary prolonged discomfort and recovery time for the athlete. �ere 
is currently a lack of evidence in the literature concerning treatment and return-to-play protocols referring speci�cally to PSP within the 
athletic community. �is case report highlights the predisposing and important factors in the history of a 34-year-old recreational male 
athlete who developed PSP. According to the knowledge of the authors, this report of PSP in a recreational athlete is the �rst of its kind 
described in South Africa. Owing to the possibility of life-threatening complications, it is important for sports physicians to be familiar with 
the important points in the history and to be made aware of the predisposing factors that may lead to PSP.

S Afr J Sports Med 2015;27(1):23-24. DOI:10.7196/SAJSM.603

Primary spontaneous pneumothorax in a recreational athlete
F J van der Col�,1,2 MB ChB; D C Janse van Rensburg,2,3 MD

1 Private Practitioner, Potchefstroom, South Africa
2 Section Sports Medicine, Faculty of Health Sciences, University of Pretoria, South Africa
3 Exercise SMART Team, University of Pretoria, South Africa

Corresponding author: F J van der Col� (medfvandercol�@yahoo.com)

Pneumothorax is defined as the presence of gas in 
the pleural cavity.[1] This can occur spontaneously 
or subsequent to direct trauma to the chest wall. 
Primary spontaneous pneumothorax (PSP) occurs in 
the absence of any underlying lung pathology,[1-3] and 

although it is a well-recognised and well-described cause of acute-
onset chest pain, the prevalence of this condition in the athletic 
community is uncommon. �is may be owing to the low incidence of 
this condition in athletes, underreporting of cases and possibly missed 
diagnoses.

Tension pneumothorax is an extremely rare but life-threatening 
complication of PSP, and if suspected, should be treated as a medical 
emergency.[2] �is case report describes a rare occurrence of PSP in 
a recreational athlete and focuses on the athlete’s medical history 
as well as the epidemiology and predisposing factors that may lead 
to PSP.

Case report
�is case involves a 34-year-old male who worked as an engineer at 
a steel production plant. He participated in a number of indoor and 
outdoor sports. In his medical history, it was noted that he su�ered 
from gastro-oesophageal re�ux disease (GERD) secondary to a hiatus 
hernia, which was diagnosed in 2009. No further medical problems 
were reported. No known allergies were reported and he did not 
smoke. He did, however, have a positive family history, with one 
female sibling having developed PSP in recent years.

Six weeks prior to the onset of his symptoms, he competed in a 
high-altitude ultra-marathon trail run in cold and wet conditions. On 
the day before his symptoms started, he was playing action cricket. He 
did not experience any symptoms and or sustain any direct trauma 
to the chest wall during the match. He presented to his general 
practitioner (GP) with complaints of acute-onset chest pain on the 
right side, which woke him during the night. �e pain progressively 

worsened the following day and did not respond to analgesic or 
antacid treatment. He did not complain of any shortness of breath, 
and attributed the pain to acid re�ux.

�e examination was noted to be unremarkable, with normal lung 
and heart sounds and normal vital signs. His GP agreed that the pain 
might be due to GERD. Antire�ux treatment was prescribed and he 
was sent home. �e pain gradually subsided over the following 5 days.

A�er 2 weeks of rest, he played another match of action cricket, but 
had to withdraw from the match because of chest pain and shortness 
of breath. A follow-up appointment with his GP was scheduled 
3 weeks a�er the initial appointment.

Immediate chest radiographs were ordered, which showed a large 
pneumothorax with 60% of the pleural space occupied by air on the 
right side of the chest (Fig. 1). No mediastinal shi� was noted on the 

Fig. 1. X-ray of lungs clearly demonstrating pneumothorax on the right.



24   SAJSM  VOL. 27  NO. 1  2015

radiograph. He was urgently referred to a cardiothoracic surgeon and 
was taken to the operating theatre the following day for a diagnostic 
bronchoscopy. �e cause of the pneumothorax was found to be a 
ruptured bulla in the apex of the right lung. �e surgeon continued 
to do a right posterolateral minithoracotomy, drained the organising 
pleural e�usion, and performed a bullectomy and complete parietal 
pleurectomy of the right lung.

�e pleura of the right lung were sent to the laboratory for histology, 
including Ziehl-Neelson staining to exclude tuberculosis infection 
and periodic acid-Schi� (PAS) staining to exclude fungal infection. 
Histology showed an eosinophylic pleuritis with reactive mesothelial 
hyperplasia. �e Ziehl-Neelson stain and PAS stain both came back 
negative. He was discharged from hospital a few days later and his 
recovery since then has been uneventful.

Discussion
Pneumothoraces can broadly be categorised as either traumatic or 
spontaneous. Spontaneous pneumothorax can further be subdivided 
into two groups: primary spontaneous pneumothorax (PSP), where 
there is no history of underlying lung pathology; and secondary 
spontaneous pneumothorax (SSP), where there is pre-existing lung 
pathology.[1-3]

PSP is more common in males, with an incidence of 7.4 - 
18 cases/100  000/year in males, and 1.2 - 6 cases/100  000/year in 
females.[2,3] Other risk factors include smoking and being a tall, thin 
male between the ages of 10 and 30 years.[3] Almost 10% of cases have 
a positive family history of PSP, which is linked to a speci�c gene 
mutation, folliculin, associated with a rare disease called Birt-Hogg-
Dubé syndrome.[3] Changes in climate, with decreased atmospheric 
pressures and colder temperatures, have been identi�ed as possible 
contributing factors in PSP.[4]

�e most common presenting symptoms of PSP are acute-onset 
pleuritic chest pain and shortness of breath, which may improve 
over a period of 24 hours.[3] Chest pain is an important and reliable 
symptom that is present in 80 - 95% of cases.[5] The physical 

examination may reveal a decrease in breath sounds on the a�ected 
side, with hyper-resonance on percussion, crepitus on palpation 
and tracheal shi�.[6] �ese signs may be very di�cult to elicit in a 
small pneumothorax, and a physician should have a high index of 
suspicion from the history.

�e threshold for ordering chest radiographs should be very low 
if a possible pneumothorax is suspected. �e majority of cases occur 
during rest, with <10% of spontaneous pneumothoraces occurring 
during exercise. �e major cause of PSP seems to be the rupture of 
bullae in the apices of one or both lungs. �e exact cause of these 
bullae still remains unclear and warrants further investigation.[2,3]

PSP seems to be a rare condition among the athlete population. 
In this case report, it is evident that the athlete had a number of risk 
factors, including sex, age, exposure to cold weather conditions and 
decreased atmospheric pressure, as well as a positive family history, all 
of which need to be taken into account when an athlete presents with 
acute-onset chest pain. �e recording of cases of PSP in the athletic 
community speci�cally is encouraged to help guide sports physicians 
to better diagnose and manage this condition. The research and 
development of safe return-to-play guidelines for these athletes are 
also recommended.

References
1. Braunwald E, Fauci AS, Kasper DL, eds. Harrison’s principles of internal medicine. 

15th ed. New York: McGraw-Hill, 2001.
2. Noppen M. Spontaneous pneumothorax: Epidemiology, pathophysiology and cause. 

Eur Respir Rev 2010;19(117):217-219. [http://dx.doi.org/10.1183/09059180.00005310] 
3. Luh S. Diagnosis and treatment of primary spontaneous pneumothorax. J Zhejiang 

Univ Sci B 2010;11(10):735-744. [http://dx.doi.org10.1631/jzus.B1000131] 
4. Haga T, Kurihara M, Kataoka H, Ebana H. In�uence of weather conditions on the 

onset of primary spontaneous pneumothorax: Positive association with decreased 
atmospheric pressure. Ann �orac Cardiovasc Surg 2013;19(3):212-215. [http://
dx.doi.org/10.5761/atcs.oa.12.01884]

5. Sik EC, Batt ME, Heslop LM. Atypical chest pain in athletes. Curr Sports Med Rep 
2009;8(2):52-58. [http://dx.doi.org/10.1249/JSR.0b013e31819c7d01] 

6. Mensinger JM. Pneumothorax in a recreational athlete. Int J Athl Ther Train
2013;18(6):27-31. 

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