Sudan Journal of Medical Sciences
Volume 13, Issue no. 2, DOI 10.18502/sjms.v13i2.2640
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Case Report

Investigation of Diffuse Hair Fall Case Due to
Vitamin D Deficiency: A Case Report and
Literature Review
H. H. Suad1 and Gad Allah Modawe2
1Omdurman Islamic university, Faculty of Medicine and Health Sciences, Department of
Dermatology
2Omdurman Islamic university, Faculty of Medicine and Health Sciences, Department of
Biochemistry

Abstract
A 45-year-old female from northern Sudan presented a complaint of diffuse hair loss
from her scalp for 4 years. The condition started when she was in Saudi Arabia, where
she resided for five years with her husband and children. The condition is associated
with fatigability and easily falling asleep. The condition is static with no known
relieving or aggravating factors. She looks well, not pale, jaundiced or cyanosed,
regular pulse, no lymph node enlargement; thyroid is central with normal size and
no lower limb edema. Scalp: looks normal, no scales or erosions. Hair: hair is dry
with normal texture; loss is diffuse, no apparent patch of alopecia. Axillary and pubic
hair is normal. All investigations were within normal values except serum vitamin
D that was found to be very low: 9 (average value 30–300). Management plans to
correct the deficiency for three months, and then a daily maintenance dose is to be
prescribed. The monitoring of vitamin D serum level is to be assessed to avoid hyper
vitaminosis. Vitamin D supplement was prescribed as oral tabs of 50,000 IU weekly for
three months. Daily maintenance dose of 1000 IU was prescribed. Improvement was
noticed starting from the first month, and excellent result was achieved after three
months of daily supplementation; daily maintenance dose was then prescribed with
an advice of being outdoors to enhance endogenous synthesis. Conclusion: Serum
vitamin D level should be assessed in patients with hair loss, especially those at a
risk because of being indoors.

Keywords: vitamin D, diffuse hair loss, scalps

How to cite this article: H. H. Suad and Gad Allah Modawe (2018) “Investigation of Diffuse Hair Fall Case Due to Vitamin D Deficiency: A Case
Report and Literature Review,” Sudan Journal of Medical Sciences, vol. 13, issue no. 2, pages 91–97. DOI 10.18502/sjms.v13i2.2640

Page 91

Corresponding Author: H. H.

Suad; email:

Dr.suadhassan4@gmail.com

Received 10 February 2018

Accepted 15 June 2018

Published 28 June 2018

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H. H. Suad and Gad Allah

Modawe. This article is

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Sudan Journal of Medical Sciences H. H. Suad and Gad Allah Modawe

1. Introduction

Vitamin D refers to a group of fat-soluble secosteroids that are very important to the

body because they increase intestinal absorption of calcium magnesium and phos-

phate, they also have other biologic effects. VitaminD3 and vitamin D2 are considered

the most important compounds in this group [1]. They can be taken in diet [2]. Vita-

min D is synthesized in the skin from cholesterol through a chemical reaction that is

dependent on sun exposure (specially UVB). It undergoes enzymatic conversion by

hydroxylation in the liver and kidney to change to the active form. They are consid-

ered as hormones rather than vitamins [3]. Cholecalciferol is converted in the liver

to 25hydroxycholecalciferol, ergocalciferol is hydroxylated in position 25 to form the

active metabolite 25 hydroxyergocalciferol, serum level of these two metabolites are

measured to assess vitamin D status of the individual [4, 5]. Calcifediol is further

hydroxylated by the kidneys to form 1,25-dihydroxycholecalciferol, the biologically

active form of vitamin D [6]. It circulates as a hormone in the blood, having a major

role in regulating the level of calcium and phosphate, they also act in enhancing the

promotion of healthy growth and bone remodeling. Calcitriol also acts as immune

modulator and anti-inflammatory [7].

Diffuse hair loss in women can be caused by many factors, the most common cause

is female pattern hair loss (FPHL) [8]. Diffuse hair loss occurs due to changes in hair

follicle cycling [9].An androgen-dependent nature has not been proven in FPHL. Most

women with FPHL do not have biochemical hyperandrogenism [1]. Women without

circulating androgens may also develop FPHL [10], suggesting a possible role for non-

androgen-dependent mechanisms. This could explain why some women with FPHL

do not respond to androgen inhibition therapy [11]. The found normal serum level of

vitamin D is needed to delay cellular-aging process in the body including hair, this fact

explains hair loss when there is vitamin D deficiency [12], and serum vitamin D level

should be assessed in women suffering from hair loss [13].

Rasheed et al. found that low serum vitamin D and serum ferritin are lower in

patients with FPHL and also diffuse hair loss in patients aging 18–45 years; they sug-

gested that serum levels of vitamin D and ferritin should be assessed, concluding that

their correction may be beneficial when they are low [14].

Cerman et al. screened 86 patients with a specific type of hair loss, alopecia areata,

in a cross-sectional study and found that vitamin D is significantly lower in patients with

alopecia areata than the control. They suggested screening all patients with alopecia

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Sudan Journal of Medical Sciences H. H. Suad and Gad Allah Modawe

areata for vitamin D deficiency and thought that supplementation of such patients with

vitamin D can be of value [15].

An endocrine society for clinical practice stated that vitamin D deficiency is common

among all age groups, and that few foods contain vitamin D, and the main source for

vitamin D is the synthesis in the skin by the ultraviolet light; so people at risk should

be screened for its deficiency because it is associated with many health problems.

The society had established guidelines for the prevention and treatment of the defi-

ciency of vitamin D and also recommended the supplementation with vitamin D for

prevention and treatment of the deficiency [16].

2. Case Report

A 45-year-old female from northern Sudan presented a complaint of diffuse hair loss

from her scalp for 4 years. The condition began when she was in Saudi Arabia, where

she resided with her husband and children for five years. The condition is associated

with fatigability and easily falling asleep. The condition is static with no known reliev-

ing or aggravating factors. Systemic review revealed no abnormality, no palpitation,

hyper- or hypohidrosis, but she feels depressed, is a newly discovered diabetic, and

hypertensiveness is unknown. She has a past history of hospital admission as a child

for tonsillectomy and adenoidectomy, but no history of blood transfusion. She’s has

no regular medications apart from Metformin 500 mg, which was prescribed by her

doctor for the control of mild diabetes mellitus. She has a family history of diabetes

mellitus and asthma, but no history of diffuses hair loss. The patient is a house wife

married for 20 years, has two daughters and two sons: the eldest is 19 years old and

the youngest is 7 years old. They live in their own house—a two-floor building with a

big courtyard in Khartoum north—but very rarely stay outside. Being a house wife she

has been staying indoors and rarely goes outdoors during the day, but when she does,

she puts on a sunscreen with SPF 50+. She has regular period, with average loss.

On examination, she looks well, not pale, jaundiced or cyanosed, regular pulse, no

lymph node enlargement, thyroid is central with normal size and no lower limb edema.

Scalp: looks normal. Hair: hair is dry with normal texture, loss is diffuse, no apparent

patch of alopecia. No scales or erosions. Axillary and pubic hair are normal.

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Sudan Journal of Medical Sciences H. H. Suad and Gad Allah Modawe

3. Investigations

Urine analysis: Normal findings; Stool analysis: Normal findings; CBC: Normal findings.

Thyroid function tests: Normal levels of T4, T3 AND TSH. Total iron load normal.

Fasting blood glucose is 121 mg/dl. HbA1c is 6. Serum vitamin D low: 9 (average value

30–300).

4. Treatment

Management plans to correct the deficiency for three months and then a daily main-

tenance dose is to be prescribed. The monitoring of vitamin D serum level is to be

assessed to avoid hyper vitaminosis.

Vitamin D supplement was prescribed as oral tabs of 50,000 IU weekly for three

months.

Daily maintenance dose of 1000 IU was prescribed.

Outcome excellent result was achieved starting from the first month in the form of

decrease in loss, and by the end of three months, the hair was normally regrowing.

The symptoms of fatigability and good mood were also improved.

5. Discussion

Vitamin D is an essential vitamin for our body serving many functions that are nec-

essary for our well-being and health. It is synthesized from a precursor in the skin

through the aid of UVB, but the body can also obtain it from diet [2].

Its role in calcium and phosphate homeostasis is well known [7], but its relation to

diffuse hair loss is still not very clear, so it is suggested by some authors to assess

the serum levels of vitamin D in females with hair loss [13]. Some studies showed an

association of vitamin D deficiency with alopecia areata, but clear studies to diffuse

hair loss are still lacking.

Diffuse hair fall is one of the common complains encountered in dermatology clin-

ics, which can be due to many factors including diseases of the scalp, for example,

Seborreic dermatitis, pityriasis capitis, etc. Some systemic and endocrine diseases and

drugs may also be involved in the etiology; in some patients, it can be due to mineral

depletion, for example, iron. Stress is also mentioned as a cause of diffuse hair loss.

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Sudan Journal of Medical Sciences H. H. Suad and Gad Allah Modawe

In this case, all the known causes of diffuse hair loss had been excluded by history,

examination and investigations.

An optimal concentration of Vitamin D is needed to delay the aging process and loss

of cells including hair [12].

Serum vitamin D in this case was measured and severe deficiency was found. Serum

vitamin D deficiency in areas like Sudan is not usually suspected because of the contin-

uous exposure to the sun since the synthesis of vitamin D in the skin requires ultraviolet

rays [3]. The deficiency in this case can be justified by the fact that the patient was

mostly living indoors in Saudi Arabia for five years. Also, the style of life in Sudan is

also changing, where there is a tendency of living in flats and houses with low sun

exposure.

The improvement in hair loss was obtained with correction of vitamin D using oral

supplements, which of course points to a close relationship between vitamin D defi-

ciency and hair loss.

So, vitamin D deficiency was found to be linked to the different modalities and types

of hair losses [14–16]; In this article, vitamin D deficiency was also found to be linked

to diffuse hair loss, but moreover correction of this deficiency and follow-up were also

done.

6. Conclusion and Recommendations

Vitamin D deficiency is to be considered in patients with diffuse hair loss.

Further studies in this respect are to be conducted.

Health education for being outdoors is necessary to avoid the deficiency of vitamin

D.

The use of sun screens should be minimized since they hinder absorption of ultravi-

olet light, and so reducing the synthesis of vitamin D in the skin leads to its deficiency

with all the consequences.

References

[1] Holick, M. F. (March 2006). High prevalence of vitamin D inadequacy and implication

for health. Mayo Clinic Proceedings, vol. 81, no. 3, pp. 353-373.

[2] Calvo, M. S., Whiting, S. J., Barton, C. N. (February 2005). Vitamin D intake: A global

perspective of current status. The Journal of Nutrition, vol. 135, no. 2, pp. 310-316.

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[3] Norman, A. W. (August 2008). From vitamin D to hormone D: Fundamentals of

the vitamin D endocrine system essential for good health. The American Journal

of Good Nutrition, vol. 88, no. 2, pp. 491S-499S, PMID 18689389.

[4] Lab Tests Online (USA). Vitamin D Tests. American Association for Clinical chemistry.

Retrieved June 23, 2013.

[5] Hollis, B. W. (January 1996). Assessment of vitamin D nutritional and hormonal

status: What to measure and how to do it. Calcified Tissue International, vol. 58,

no. 1, pp. 4-5.

[6] Holick, M. F., Schnoes, H. K., DeLuca, H. F., et al. (July 1971). Isolation and

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intestine. Biochemistry, vol. 10, no. 14, pp. 2799-2804.

[7] NIH Office of Dietary Supplements. (February 11, 2016). Vitamin D. Retrieved June

6, 2017.

[8] Olsen, E. A. (2001). Female pattern hair loss. Journal of the American Academy of

Dermatology, vol. 45, pp. S70-S80.

[9] Tobin, D. J., Guinn, A., Magerl, M., et al. (2003). Plasticity and cytokinetic dynamics

of the hair follicle mesenchyme: Implications for hair growth control. Journal of

Investigative Dermatology, vol. 120, pp. 895-904.

[10] Orme, S., Cullen, D., and Messenger, A. (1999). Diffuse female hair loss: Are

androgens necessary? British Journal of Dermatology, vol. 141, pp. 521-523.

[11] Olsen, E. (2003). Female pattern hair loss, in E. Olsen (ed.) Disorders of Hair Growth:

Diagnosis and Treatment (2nd edition), pp. 338-339. New York, NY: McGraw-Hill

Professional.

[12] Bolland, M. J., Ames, R. W., Grey, A. B., et al. (2008). Does degree of baldness

influence vitamin D status? The Medical Journal of Australia, vol. 189, nos. 11-12,

pp. 674-675.

[13] Amor, K., Rashid, R., and Mirmirani, P. (2010). Does D matter? The role of vitamin D

in hair disorders and hair follicle cycling. Dermatology Online Journal, vol. 16, no. 2,

p. 3.

[14] Rasheed, H., Mahjoob, D., Hegazy, R., et al. (2013). Serum ferritin and vitamin D in

female hair loss: Do they play a role [Abstract]? Skin Pharmacology and Physiology,

vol. 26, no. 2, pp. 101-107.

[15] Cerman, A. A., Solak, S. S., and Altunay, l. K. (March 2014). Vitamin D deficiency in

alopecia areata. British Journal of Dermatology, vol. 170, no. 6, pp. 1299-1304.

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[16] Holick, M. F., Binkley, N. C., Bischoff-Ferrari, H. A., et al. (July 1, 2011). Evaluation,

treatment, and prevention of vitamin D deficiency: An Endocrine Society clinical

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7, pp. 1911-1930.

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	Introduction
	Case Report
	Investigations
	Treatment
	Discussion
	Conclusion and Recommendations
	References