1Department of Pediatrics, Sarojini Naidu Children Hospital, Moti Lal Nehru Medical College, Allahabad, India; 2Department of Medicine, Central 
Research Institute of Unani Medicine, Hyderabad, India
*Corresponding Author’s e-mail: sha.akht@yahoo.com

مرض "كايف" يف سن الرَّضاع
معضلة تشخيصية ألطباء الرعاية الصحية األولية

�صاهد اأخ�رش �صدقي، جوليناز فاطمة �صدقي، ماني�صا موريا، اأنباها �رشيفازتافا، موك�س فري �صينق

abstract: Caffey disease is a rare and self-limiting condition characterised by cortical hyperostosis with inflammation 
of adjacent fascia and muscles. It usually presents in infancy and clinical features include hyperirritability, acute 
inflammation with swelling of overlying soft tissues and subperiosteal new bone formation. Awareness of the existence 
of this rare condition and its typical clinical and radiological profile will avoid unnecessary investigations and treatment 
and help the physician to explain its good prognosis to parents of affected children. We report a three-month-old male 
infant who presented to the Outpatient Paediatrics Department at Moti Lal Nehru Medical College, Allahabad, India, 
in 2018 with a right shoulder mass, decreased upper limb movements and irritability. The patient was treated with 
ibuprofen and paracetamol. Irritability and limitation of movement improved over a treatment period of two weeks.

Keywords: Caffey Disease; Infant; Prostaglandin E1; Thrombocytosis; Case Report; India.

دوُد ذاِتيَّا، ويتميز بفرط تعظم ق�رشي، والتهاب اللفافات والع�صالت املجاورة، ويظهر  امللخ�ص: مر�س "كايف" مر�س نادر احلدوث وحَمْ
حدوث  واإدراك  ال�صمحاق.  حتت  اجلديدة  العظام  وتكوين  الرخوة،  الفوقية  الأن�صجة  بتورم  م�صحوبا  حاد  والتهاب  تهيج،  كفرط  اكلينيكيا 
مثل هذه احلالة النادرة واأعرا�صها الإكلينيكية والإ�صعاعية يجنبنا عمل كثري من الفحو�صات والعالجات غري ال�رشورية، وي�صاعد الطبيب 
اأح�رش  اأ�صهر،  ثالثة  عمره  ذكر  ر�صيع  حالة  هنا  ون�صجل  املاآل.  جيد  املر�س  باأن  وتطمينهما  املري�س  الطفل  دي  لوال  احلالة  �رشح  على 
لعيادة الأطفال اخلارجية يف كلية "موتي لل نهرو" الطبية باهلل اآباد بالهند يف عام 2018م، وهو م�صاب بكتلة يف كتفه الأمين قللت من 
حركة طرفه الأمين، و�صببت له تهيجا. ومت عالج الر�صيع بدوائي ابيوبروفني وبرا�صيتامول. حت�صنت بعد اأ�صبوعني من العالج حالة التهيج 

وحمدودية احلركة.
الكلمات املفتاحية: مر�س كايف؛ ر�صيع؛ برو�صتاغالندين إي 1؛ ازدياد ال�صفيحات الدموية؛ تقرير حالة؛ الهند.

Caffey Disease in Infancy
A diagnostic dilemma for primary care physicians

*Shahid A. Siddiqui,1 Gulnaz F. Siddiqui,2 Manisha Maurya,1 Anubha Shrivastava,1 Mukesh V. Singh1

Sultan Qaboos University Med J, February 2020, Vol. 20, Iss. 1, pp. e109–111, Epub. 9 Mar 20
Submitted 26 May 19
Revision Req. 2 Jul 19; Revision Recd. 30 Aug 19
Accepted 14 Nov 19

Caffey disease or infantile cortical hyper-ostosis (ICH) is a rare disorder characterised by acute inflammation of the periosteum and 
the overlying soft tissues and is usually accompanied 
by systemic changes of fever and irritability. It usually 
presents in infancy and clinical features include 
sudden onset of irritability, fever, decreased appetite 
and swelling of overlying soft tissues that precedes 
cortical thickening of underlying bones.1 We report a 
case of Caffey disease to emphasise that, although rare, 
Caffey disease should be kept in mind in the diagnostic 
approach to childhood bony swellings.

Case Report

A three-month-old afebrile male infant presented to 
the Outpatient Paediatrics Department at Moti Lal 
Nehru Medical College, Allahabad, India, in 2018 with 
a history of mild irritability, a right shoulder mass and 
decreased upper extremity movement for the previous 
two weeks. There were no symptoms associated with 

the respiratory, gastrointestinal or urinary systems. 
He was born full-term at 39 completed weeks of gest- 
ation via normal vaginal delivery with a birth weight 
of 2.5 kg, a length of 49 cm and a head circumference 
of 34.5 cm. His Apgar scores were 6 and 8 at 1 and 
5 minutes, respectively. The infant was vaccinated 
according to his age with no significant antenatal or 
postnatal history. There was no history of prosta- 
glandin E1 infusion to maintain ductal patency during 
the neonatal period. He was the first child of the 
couple and there was no family history of similar 
complaints. The infant was exclusively breast-fed since 
birth with no vitamin supplementation. There was no 
history of trauma, intramuscular injections, lethargy 
or convulsions. Child abuse was not suspected as 
he was the outcome of a planned pregnancy. Upon 
examination, the patient was active and alert, afebrile, 
mildly irritable and had no dysmorphic facial features. 
Anthropometric examination was normal for his age. 
A right shoulder mass with soft tissue swelling over 
the body of scapula was noted [Figure 1]. The infant 

This work is licensed under a Creative Commons Attribution-NoDerivatives 4.0 International License.

https://doi.org/10.18295/squmj.2020.20.01.017

case report

https://creativecommons.org/licenses/by-nd/4.0/


Caffey Disease in Infancy 
A diagnostic dilemma for primary care physicians

e110 | SQU Medical Journal, February 2020, Volume 20, Issue 1

had decreased upper extremity movements with pain 
during passive movement of the right upper extremity. 
However, deep tendon reflexes were well-preserved 
and there was no swelling or tenderness over other 
bones. The remainder of the systemic examination 

was within normal limits. X-ray examination of the 
affected shoulder revealed diffuse thick irregular ossi- 
fication along the cortex of the scapula involving both 
anterior and posterior surfaces with diffuse thickening 
of the scapula with a thickened cortex [Figure 2]. 
Ultrasonography of the shoulder showed that the 
adjacent muscles around the scapula were relatively 
bulky with altered echogenicity without any evidence 
of fluid collections [Figure 3]. Biochemical and 
haematological investigations were mostly unremark- 
able except for an elevated C-reactive protein level 
[Table 1]. Genetic analysis could not be done as it was 
not available at the centre and the parents refused 
further investigations as it is a self-limiting condition.

On the basis of the clinical findings, elevated 
C-reactive protein and radiology, the infant was 
diagnosed with Caffey disease. Subsequently, he was 
treated with ibuprofen and paracetamol. Irritability 
and limitation of movement improved over a treatment 
period of two weeks. He was doing well on follow-up.

Discussion

Caffey disease is a rare, self-limiting disorder of early 
infancy with an onset within the first six months of 
life and usually resolves without sequelae by two years 
of age. It is often sporadic with occasional reports 
of being autosomal dominant or recessive.2 It is charact- 
erised by a triad of systemic symptoms (e.g. irritability 
and fever), soft tissue swelling and underlying cortical 
bone thickening.3,4 The mandible is the most commonly 
affected site followed by scapula, clavicle, ribs and long 
bones. 

Laboratory findings include elevated erythrocyte 
sedimentation rate and in some cases high alkaline 
phosphatase, thrombocytosis, anaemia and elevated 

 
Figure 1: Photograph of the right shoulder of a three-
month-old male infant showing a mass with normal skin 
overlying the mass.

 
Figure 2: X-ray images of the right shoulder of a three-
month-old male infant showing diffuse scapular periosteal 
reaction with subperiosteal new bone formation.

 
Figure 3: Ultrasonography of the right shoulder of a three-month-old male infant showing relatively bulky adjacent 
muscles around the scapula with altered echogenicity without any evidence of fluid collections.



Shahid A. Siddiqui, Gulnaz F. Siddiqui, Manisha Maurya, Anubha Shrivastava and Mukesh V. Singh

Case Report | e111

immunoglobulin levels.5 There are no specific labor- 
atory tests for Caffey disease. Radiology is the most 
valuable tool in diagnosing Caffey disease as it shows 
layers of periosteal new bone formation with cortical 
thickening.

However, its diagnosis may be delayed as it 
mimics many other conditions such as osteomyelitis, 
hypervitaminosis A, syphilis, scurvy, bone tumours, 
prolonged prostaglandin E infusion and child abuse.6 
As the current patient was thriving and there was an 
absence of early bone radiological signs and a normal 
white blood cell count, osteomyelitis was ruled out. 
There was no history of vitamin A supplementation 
as national programmes in India recommend routine 
vitamin A supplementation from nine months onwards. 
Negative venereal disease research laboratory tests for 
both the mother and infant made syphilis an unlikely 
diagnosis. Malignancy and scurvy were also excluded 
based on normal radiography/ultrasonography and 
haematological tests. In addition, the infant did not 

have history of prostaglandin infusion during the 
neonatal period to maintain ductal patency.

After excluding various differential diagnoses 
and given the characteristic features of Caffey disease 
that were present in the current case (irritability, a 
right shoulder mass and decreased upper extremity 
movements with periosteal new bone formation), the 
patient was diagnosed with Caffey disease.

Conclusion

Awareness of the existence of Caffey disease and its 
typical clinical and radiological profile as well as its self-
limiting nature will avoid unnecessary investigations 
and treatment in resource-limited countries such as 
India.

References
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understood or unknown. In: Kliegman RM, Stanton BF, St 
Geme III JW, Schor NF, Eds. Nelson Textbook of Pediatrics. 1st 
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2. Nistala H, Mäkitie O, Jüppner H. Caffey disease: New pers- 
pectives on old questions. Bone 2014; 60:246–51. https://doi.
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3. Kutty N, Thomas D, George L, John TB. Caffey disease or 
infantile cortical hyperostosis: A case report Oman Med J 2010; 
25:134–6. https://doi.org/10.5001/omj.2010.36.

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Table 1: Biochemical findings of a three-month-old infant 
with Caffey disease

Investigation Finding Normal 
range

Haemoglobin in g/dL 14.2 10.5–14.0

Total WBC count × 104/µL 1.12 0.6–1.4

Platelet cell count × 105/µL 2.2 1.5–4.0

Serum urea in mg/dL 21 20–35

Creatinine in mg/dL 0.68 0.20–0.50

C-reactive protein in mg/dL 1.2 0.08–1.12

VDRL test Negative* -

WBC = white blood cell; VDRL = venereal disease research laboratory.
*The mother also tested negative.

https://doi.org/10.1016/j.bone.2013.12.030
https://doi.org/10.1016/j.bone.2013.12.030
https://doi.org/10.5001/omj.2010.36
https://doi.org/10.1007/s00247-010-1869-2
https://doi.org/10.1007/s12098-008-0027-4
https://doi.org/10.1007/s13312-017-1023-4