Urological Oncology

26 Urology Journal    Vol 7    No 1    Winter 2010

Clinical Relevance of HER-2/neu Overexpression 
in Patients With Testicular Nonseminomatous germ 
Cell Tumor
Mohammad Reza ghavamnasiri,1 Hamid Saeedi Saedi,1 Soodabeh Shahid Sales,1 
Kamran ghafarzadegan2

Introduction: Recent scientific attention has focused on the role of growth 
factors in the progression of cancer. HER-2/neu is an epidermal growth 
factor receptor that is demonstrated to have correlation with poor prognosis 
of many cancers. This study evaluated the overexpression of HER-2/neu 
protein and its clinical importance in nonseminomatous germ cell tumors of 
the testis.
Materials and Methods: Testis specimens of 54 patients with testicular 
nonseminomatous germ cell tumors, referred to Omid Hospital from 2001 
to 2007, were re-evaluated and the patients’ records were reviewed. Patients’ 
age, tumor subtype, tumor stage, tumor markers, therapeutic response, and 
disease-free survival were assessed and the specimens were evaluated for the 
degree of HER-2/neu expression using an immunohistochemistry method.
Results: Immunohistochemical staining was performed for 54 specimens. 
Overexpression of HER-2/neu was seen in 33.3% of the patients with 
nonseminomatous germ cell tumors, especially in those with teratocarcinoma 
subtype compared to those with mixed germ cell tumors or embryonal 
cell carcinoma. However, HER-2/neu overexpression did not show any 
correlation with tumor stage, therapeutic response, disease-free survival, age, 
β-human chorionic gonadotropin, or α-fetoprotein.
Conclusion: We observed overexpression of HER-2/neu receptor in 
teratocarcinoma subtype of germ cell tumor. We suggest further studies to 
evaluate the clinical importance of this finding.

Urol J. 2010;7:26-9. 
www.uj.unrc.ir

Keywords: germ cell and embryonal 
neoplasms, neoplasm proteins, 

prognosis, tumor markers

1Department of Radiation Oncology, 
Mashhad University of Medical 

Sciences, Mashhad, Iran
2Department of Pathology, Mashhad 

University of Medical Sciences, 
Mashhad, Iran

Corresponding Author:
Soodabeh Shahidsales, MD

Cancer Research Center, Omid 
Hospital, Koohsangi St,  

Mashhad, Iran
Tel: +98 915 316 0721

Fax: +98 511 846 1518
E-mail: soodabehshahidsales@gmail.com

Received May 2009
Accepted August 2009

INTRODUCTION
Recent scientific attention has 
focused on the role of growth 
factors in the progression 
of cancer. HER-2/neu is an 
epidermal growth factor receptor 
(EGFR)—encoded by the ERBB2 
gene (formerly named HER-2/
neu)—that is overexpressed on 
the cell surface of approximately 
25% to 30% of breast cancers.(1) 
This expression correlates with 

a relatively poor prognosis for 
patients with breast cancer; it is 
associated with a shorter disease-
free survival and overall survival. (2) 
HER-2/neu has proved to be a 
useful therapeutic target in many 
cancers. Treatment of patients 
with HER-2/neu-amplified tumors 
with trastuzumab, a monoclonal 
antibody, results in a better clinical 
response rate.(3) Overexpression of 
HER-2/neu has been reported in 



HER-2/neu Overexpression in germ Cell Tumor—ghavamnasiri et al

27Urology Journal    Vol 7    No 1    Winter 2010

many epithelial malignancies, including cancers 
of the lung, prostate, bladder, pancreas, and 
esophagus, as well as sarcoma.(4-13) However, there 
is no evidence so far that HER-2/neu expression 
is of prognostic relevance in these malignancies.

The potential role of HER-2/neu in germ cell 
tumors (GCTs) is unknown. Some recent studies 
have tested these tumors immunohistochemically 
with monoclonal anti-HER-2/neu antibody. (14) 
The objective of our study was to determine 
the clinical importance of HER-2/neu protein 
overexpression in GCTs.

MATERIALS AND METHODS
We studied 54 patients with documented 
testicular nonseminomatous GCT referred 
to Omid Hospital between 2001 and 2007. 
Pathology specimens were collected and the 
patients’ records were reviewed. Patients’ age, 
tumor subtype, tumor stage, therapeutic response, 
tumor markers, and disease-free survival were 
assessed and the specimens were evaluated 
for the degree of HER-2/neu expression by 
immunohistochemistry methods (HercepTest kit, 
Dako, Carpinteria, California, USA), according 
to the manufacturer’s instructions. A pathologist 
who was unaware of tumor type evaluated all 
immunohistochemistry specimens. The staining 
intensity was scored from 0 to 3+ using the breast 
cancer HER-2/neu scoring system,(1) with 2+ 
or 3+ staining considered positive for protein 
overexpression.

Patients with and without HER-2/neu 
overexpression were compared in terms of tumor 
characteristics, outcome, and tumor markers 
using the t test and the chi-square test, where 
appropriate. Continuous variables were shown as 
mean ± standard deviation. P values less than .05 
were considered significant.

RESULTS
Immunohistochemical staining was performed in 
54 testis specimens of patients with documented 
nonseminomatous GCT. Results of the test are 
shown in Table 1. HER-2/neu overexpression 
was reported in 18 specimens (33.3%). A positive 
overexpression was more prominent in cases of 

teratocarcinoma, but stage of the tumor and the 
response to treatment were not linked with HER-2 
/neu expression (Table 2). The frequency of 
HER-2/neu overexpression in tumor subtypes of 
different stages is demonstrated in Table 3. HER-2 
/neu overexpression was associated with a lower 
level of lactate dehydrogenase (P = .006), but it 
was not linked with the other tumor markers 
(Table 4). 

HER-2/neu Expression Patient (%)
Negative 15 (27.8)
1+ 21 (38.9)
2+ 15 (27.8)
3+ 3 (5.6)

Table 1. HER-2/neu Expression in Patients With 
Nonseminomatous germ Cell Tumor of Testis

HER-2/neu Overexpression
Tumor Stage No Yes

Embryonal cell
I 1 0
II 8 0
III 3 1

Teratocarcinoma
I 1 3
II 2 1
III 0 2

Mixed tumor
I 8 4
II 8 6
III 4 2

Table 3. Relation of HER-2/neu Overexpression With Stage of 
Tumor Subtype in Patients With Nonseminomatous germ Cell 
Tumor of Testis

HER-2/neu Overexpression
Variable No Yes P

Tumor subtype
Embryonal cell 12 (92.3) 1 (7.7)
Teratocarcinoma 3 (33.3) 6 (66.7)
Mixed tumor 21 (65.6) 11 (34.4) .02

Tumor stage
I 11 (61.1) 7 (38.9)
II 19 (76.0) 6 (24.0)
III 6 (54.5) 5 (45.5) .38

Response to treatment
Cure 21 (63.6) 12 (36.4)
Recurrence 8 (72.7) 3 (27.3) .72

*Values in parentheses are percents.

Table 2. Relation of HER-2/neu Overexpression With Tumor 
Characteristics and Outcome of Treatment in Patients With 
Nonseminomatous germ Cell Tumor of Testis*



HER-2/neu Overexpression in germ Cell Tumor—ghavamnasiri et al

28 Urology Journal    Vol 7    No 1    Winter 2010

Follow-up duration ranged from 2 to 91 months. 
The mean disease-free survival time was not 
significantly different between the patients 
with and without HER-2/neu overexpression 
(Table 4).

DISCUSSION
The recent availability of targeted therapy with 
tyrosine kinase inhibitors, particularly with 
agents directed against EGFR, offers new hope for 
effective and better tolerated therapy for human 
neoplasms. We designed a preliminary study 
evaluating the expression of HER-2/neu among 
nonseminomatous GCTs. 

The expression of EGFR has been previously 
evaluated in primary GCTs. Shuin and associates 
reported the expression of EGFR at the 
transcriptional level in 2 of 3 immature teratomas, 
but no expression could be demonstrated in 
15 seminomas and 6 embryonal carcinomas.(15) 
Moroni and colleagues evaluated the expression 
of EGFR by immunohistochemistry in a 
series of 24 testicular tumors. The expression 
of EGFR appeared to be restricted to the 
β-human chorionic gonadotropin (β-HCG)-
positive component (choriocarcinoma) in 16 
of 18 primary nonseminomatous GCTs. In 
contrast, 1 Leydig cell tumor, 5 seminomas, and 
β-HCG-negative components of GCTs did not 
express EGFR.(16) Recently, Kollmannsberger 
and coworkers evaluated the expression 
of EGFR by immunohistochemistry in a 
series of 22 patients with platinum-resistant 
GCTs and 12 patients with chemosensitive 
GCTs. They reported that the presence of 
EGFR was restricted to trophoblastic giant 
cells and the syncytiotrophoblastic elements 
of 4 nonseminomas, including 1 pure 
choriocarcinoma, and to a secondary non-germ-
cell malignancy arising from a transformed 

teratoma. There were no differences in the 
pattern of EGFR expression between platinum-
resistant and platinum-sensitive patients.(17) 

In the present study, we found overexpression 
of HER-2/neu in 33.3% of the patients with 
nonseminomatous GCTs, especially in those 
with teratocarcinoma subtype. Our experience 
with agents targeting EGFR did not show any 
correlation between tumor stage or therapeutic 
response and the degree of expression of HER-2/
neu. The mean disease-free survival, age, β-HCG, 
and α-fetoprotein were almost similar between the 
two groups of patients with and without HER-2/ 
neu overexpression. In our study and several 
other previous investigations, the overexpression 
of HER-2/neu in teratocarcinoma was more 
frequently observed compared to mixed GCTs 
or embryonal cell carcinoma. Mandoky and 
colleagues also studied clinical relevance of HER-2 
/neu expression in germ cell testicular tumors 
in 2004, and they reported that teratomatous 
and choriocarcinoma components showed 
significantly higher HER-2/neu expression 
compared to other histological subtypes of 
GCTs.(18) Mandoky and colleagues also studied 
expression of HER-2/neu in testicular tumors in 
2003 and showed that overexpression of HER-2/ 
neu was restricted to the more differentiated 
histotypes.(19) 

In our cohort, there was no significant correlation 
between expression of HER-2/neu and β-HCG, 
α-fetoprotein, patient survival, or age. However, 
some investigators such as Soule and colleagues 
who studied HER-2/neu expression in GCTs 
have reported that overexpression of the HER-2/ 
neu protein in GCTs is of prognostic or 
therapeutic relevance.(20) Mandoky and colleagues 
also reported that HER-2/neu overexpression was 
associated with an adverse clinical outcome and 
had a prognostic role in testicular GCTs.(18) Some 

HER-2/neu Overexpression
Parameter No Yes P

Disease-free, mo 20.77 ± 32.09 24.94 ± 22.28 .58
Age, y 28.47 ± 9.21 25.83 ± 11.90 .37
β-human chorionic gonadotropin, mU/mL 1505.49 ± 7672.56 240.35 ± 373.76 .49
α-fetoprotein, ng/mL 184.37 ± 341.88 441.38 ± 1268.53 .26
Lactate dehydrogenase, U/L 658.77 ± 999.80 253.44 ± 633.26 .006

Table 4. Disease-free Survival Time, Age, and Tumor Markers in Patients With and Without HER-2/neu Overexpression



HER-2/neu Overexpression in germ Cell Tumor—ghavamnasiri et al

29Urology Journal    Vol 7    No 1    Winter 2010

other studies showed the correlation between 
HER-2/neu overexpression and histological 
subtype (teratocarcinoma), but they could not 
demonstrate the exact clinical importance of this 
finding. The reason might be the small sample 
volume that would limit further interpretations 
about clinical factors.(14) Thus, we suggest 
further investigations with more cases and with 
prolonged follow-up to evaluate clinical role 
of HER-2/neu expression in predicting disease 
course and treatment outcome. We suggest studies 
especially focused on teratocarcinoma with 
overexpression of HER-2/neu receptor in order 
to find a practical and clinical predictor factor for 
treatment planning.

CONCLUSION
We observed overexpression of HER-2/neu 
receptor in teratocarcinoma subtype of GCT. 
We suggest further studies to evaluate the clinical 
importance of this finding.

CONFLICT OF INTEREST
None declared.

REFERENCES
1. Slamon DJ, Clark gM, Wong Sg, et al. Human 

breast cancer: correlation of relapse and survival with 
amplification of the HER-2/neu oncogene. Science. 
1987;235:177-82.

2. Slamon DJ, godolphin W, Jones LA. Studies of the 
HER-2/neu proto-oncogene in human breast and 
ovarian cancer. Science. 1989;244:707-12.

3. Vogel CL, Cobleigh M, Tripathy D. Superior outcomes 
with herceptin (trastuzumab) (H) in fluorescence in situ 
hybridization (FISH)-selected patients. Am Soc Clin 
Oncol. 2001;20:86.

4. Laptalo L, Lara PN Jr, Lau DH, et al. HER-2/neu 
screening in advanced non-small cell lung cancer 
(NSCLC): a California cancer consortium trial of 
trastuzumab and docetaxel. Am Soc Clin Oncol. 
2001;20:335a.

5. gray CR, Lara PN, Jr, gandara D, et al. HER-2/
neu screening in hormone refractory prostate cancer 
(HRPC): a California cancer consortium trial of 
trastuzumab and docetaxel. Am Soc Clin Oncol. 
2001;20:181a. 

6. Reese DM, Small EJ, et al. HER-2 expression in 
androgen-independent prostate cancer. Am Soc Clin 
Oncol. 2000;19:347a. 

7. Jimenez RE, grignon DJ, Vaishampayan U, et al. 
Analysis of HER-2/neu overexpression in primary and 
metastatic transitional cell carcinoma of the bladder. 
Am Soc Clin Oncol. 2000;19:329a.

8. Estrada CR, Coogan CL, Kapur S, et al. HER-2/neu 
receptor protein over-expression in grade I, II and 
III bladder transitional cell carcinoma. Am Soc Clin 
Oncol. 2001;20:199a.

9. Safran H. HER-2/neu overexpression in pancreatic 
adenocarcinoma. Am Soc Clin Oncol. 2000;19:317a.

10. Safran H, Ramanathan R, Schwartz J, et al. Herceptin 
and gemcitabine for metastatic pancreatic cancers 
that overexpress HER-2/neu. Am Soc Clin Oncol. 
2001;20:130a.

11. Steinhoff M, Tantravahi U, King T, et al. HER-2/
neu overexpression in adenocarcinoma of the distal 
esophagus. Am Soc Clin Oncol. 2001;20:150b. 

12. Tsai JY, Aviv H, Benevenia J, et al. Prognostic factors 
in osteosarcoma—role of HER-2/neu and p53: an 
immunohistochemical (IHC) and fluorescence in situ 
hybridization (FISH) analysis. Am Soc Clin Oncol. 
2001;20:295b.

13. Oliveira AM, Medeiros F, Okuno SH, et al. HER-2/neu 
protein overexpression is a rare event in adult soft 
tissue sarcomas. Am Soc Clin Oncol. 2001;20:294b.

14. Henley J, Einhorn L. C-ERB-2 (HER-2/neu) 
overexpression in recurrent germ cell tumors. Am Soc 
Clin Oncol. 1999;18:341a.

15. Shuin T, Misaki H, Kubota Y, et al. Differential 
expression of protooncogenes in human germ-cell 
tumors of the testis. Cancer. 1994;73:1721-7. 

16. Moroni M, Veronese S, Schiavo R, et al. Epidermal 
growth factor receptor expression and activation in 
nonseminomatous germ-cell tumors. Clin Cancer Res. 
2001;7:2770-5.

17. Kollmannsberger C, Mayer F, Pressler H, et al. 
Absence of c-KIT and members of the epidermal 
growth factor receptor family in refractory germ-cell 
cancer. Cancer. 2002;95:301-8.

18. Mándoky L, géczi L, Bodrogi I, Tóth J, Csuka O, 
Kásler M. Clinical relevance of HER-2/neu expression 
in germ-cell testicular tumors. Anticancer Res. 
2004;24:2219-24.

19. Mandoky L, geczi L, Bodrogi I, Toth J, Bak M. 
Expression of HER-2/neu in testicular tumors. 
Anticancer Res. 2003;23:3447-51.

20. Soule S, Baldridge L, Kirkpatrick K, et al. HER-2/
neu expression in germ cell tumours. J Clin Pathol. 
2002;55:656-8.