id	author	title	date	pages	extension	mime	words	sentence	flesch	summary	cache	txt
cord-325192-italbsed	Desai, Tanay M.	IFITM3 Restricts Influenza A Virus Entry by Blocking the Formation of Fusion Pores following Virus-Endosome Hemifusion	2014-04-03		.txt	text/plain	8298	345	46	hemifusion to both red blood cell and planar bilayer membranes Viral Membrane Fusion and Nucleocapsid Delivery into the Cytoplasm are Distinct Events in Some Flaviviruses Observation of single influenza virus-cell fusion and measurement by fluorescence video microscopy Diffusion and redistribution of lipid-like molecules between membranes in virus-cell and cell-cell fusion systems Membrane flux through the pore formed by a fusogenic viral envelope protein during cell fusion The pathway of membrane fusion catalyzed by influenza hemagglutinin: restriction of lipids, hemifusion, and lipidic fusion pore formation Singleparticle kinetics of influenza virus membrane fusion Fusion of Mature HIV-1 Particles Leads to Complete Release of a Gag-GFP-Based Content Marker and Raises the Intraviral pH Cellular mechanism of U18666A-mediated apoptosis in cultured murine cortical neurons: bridging Niemann-Pick disease type C and Alzheimer's disease Lipid and cholesterol trafficking in NPC Ebola virus entry requires the cholesterol transporter Niemann-Pick C1 Small molecule inhibitors reveal Niemann-Pick C1 is essential for Ebola virus infection Regulation of the V-ATPase along the endocytic pathway occurs through reversible subunit association and membrane localization pH-dependent hemolysis by influenza, Semliki, Forest virus, and Sendai virus The transport of low density lipoprotein-derived cholesterol to the plasma membrane is defective in NPC1 cells Multiple cationic amphiphiles induce a Niemann-Pick C phenotype and inhibit Ebola virus entry and infection Cholesterol promotes hemifusion and pore widening in membrane fusion induced by influenza hemagglutinin Sterols and sphingolipids strongly affect the growth of fusion pores induced by the hemagglutinin of influenza virus Multiphasic effects of cholesterol on influenza fusion kinetics reflect multiple mechanistic roles Non-vesicular lipid transport by lipid-transfer proteins and beyond Endosome-tocytosol transport of viral nucleocapsids Old world arenaviruses enter the host cell via the multivesicular body and depend on the endosomal sorting complex required for transport Imaging single retrovirus entry through alternative receptor isoforms and intermediates of virus-endosome fusion The lipid-anchored ectodomain of influenza virus hemagglutinin (GPI-HA) is capable of inducing nonenlarging fusion pores Restricted movement of lipid and aqueous dyes through pores formed by influenza hemagglutinin during cell fusion Inhibition of HIV-1 endocytosis allows lipid mixing at the plasma membrane, but not complete fusion Characterization of Lassa virus cell entry and neutralization with Lassa virus pseudoparticles Comprehensive analysis of ebola virus GP1 in viral entry Multifaceted mechanisms of HIV-1 entry inhibition by human alphadefensin We wish to thank Dr. David Steinhauer (Emory University) for the gift of R2376 antibody, Dr. Laura Liscum (Tufts University) for CHO-NPC1 2 cells, Dr. F.-L. Cosset (UniversitÃ© de Lyon, France) and Dr. L. Rong (University of Illinois) for Lassa virus and Ebola virus GP-expressing vectors, respectively. HIV enters cells via endocytosis and dynamin-dependent fusion with endosomes An enzymatic virus-like particle assay for sensitive detection of virus entry Membrane hemifusion: crossing a chasm in two leaps The energetics of membrane fusion from binding, through hemifusion, pore formation, and pore enlargement Visualizing infection of individual influenza viruses Characterization of the early events in dengue virus cell entry by biochemical assays and single-virus tracking GPI-anchored influenza hemagglutinin induces	cache/cord-325192-italbsed.txt	txt/cord-325192-italbsed.txt
