id	author	title	date	pages	extension	mime	words	sentence	flesch	summary	cache	txt
cord-302616-1uwrcvjx	Steenblock, Charlotte	Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and the neuroendocrine stress axis	2020-05-07		.txt	text/plain	3877	139	33	On the use of corticosteroids for 2019-nCoV pneumonia Renin-angiotensin-aldosterone system inhibitors in patients with Covid-19 Timely mental health care for the 2019 novel coronavirus outbreak is urgently needed Developmental trajectories of early life stress and trauma: a narrative review on neurobiological aspects beyond stress system dysregulation The adrenal gland in stress -adaptation on a cellular level Hypothalamic-pituitary-adrenal and hypothalamic-pituitary-gonadal axes: sex differences in regulation of stress responsivity Sex differences and stress across the lifespan SARS-CoV-2 cell entry depends on ACE2 and TMPRSS2 and is blocked by a clinically proven protease inhibitor Structure, function, and antigenicity of the SARS-CoV-2 spike glycoprotein Origin and evolution of the 2019 novel coronavirus The renin-angiotensin-aldosterone system and its suppression Novel therapeutic approaches targeting the Renin-Angiotensin System and associated peptides in hypertension and heart failure The vasoprotective axes of the reninangiotensin system: Physiological relevance and therapeutic implications in cardiovascular, hypertensive and kidney diseases The ACE2/Angiotensin-(1-7)/MAS axis of the Renin-Angiotensin System: focus on angiotensin Classical Renin-Angiotensin system in kidney physiology Are patients with hypertension and diabetes mellitus at increased risk for COVID-19 infection? Neutralization of SARS-CoV-2 spike pseudotyped virus by recombinant ACE2-Ig Increasing brain angiotensin converting enzyme 2 activity Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) and the neuroendocrine stress axis decreases anxiety-like behavior in male mice by activating central Mas receptors Coupling corticotropin-releasing-hormone and angiotensin converting enzyme 2 dampens stress responsiveness in male mice Lethal infection of K18-hACE2 mice infected with severe acute respiratory syndrome coronavirus Severe acute respiratory syndrome coronavirus infection causes neuronal death in the absence of encephalitis in mice transgenic for human ACE2 Mice transgenic for human angiotensin-converting enzyme 2 provide a model for SARS coronavirus infection Single-cell RNA-seq data analysis on the receptor ACE2 expression reveals the potential risk of different human organs vulnerable to 2019-nCoV infection Effects of Taurine on ACE, ACE2 and HSP70 expression of hypothalamicpituitary-adrenal axis in stress-induced hypertensive rats Organ distribution of severe acute respiratory syndrome (SARS) associated coronavirus (SARS-CoV) in SARS patients: implications for pathogenesis and virus transmission pathways The origin, transmission and clinical therapies on coronavirus disease 2019 (COVID-19) outbreak -an update on the status Multiple organ infection and the pathogenesis of SARS The neuroinvasive potential of SARS-CoV2 may play a role in the respiratory failure of COVID-19 patients COVID-19: the gendered impacts of the outbreak COVID-19 and Italy: what next? An appropriate response during acute stress is imperative for survival, but frequent or prolonged activation can change the functional tone of stress systems with ensuing deleterious effects.	cache/cord-302616-1uwrcvjx.txt	txt/cord-302616-1uwrcvjx.txt
